Dyspareunia pathophysiology: Difference between revisions
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==Pathophysiology== | ==Pathophysiology== | ||
Sexual pain disorders can therefore be considered as | Sexual pain disorders can therefore be considered as:<ref> 10.1007/978-3-319-52539-6_20</ref> | ||
*multifactorial | *multifactorial | ||
*multisystemic | *multisystemic | ||
Revision as of 00:21, 21 September 2020
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Overview
Dyspareunia is caused by medical causes, psychological problems, and pathophysiology can be considered as multifactorial, multisystemic or complex. dyspareunia vary based on the underlying causes . For example, estrogen deficiency causes atrophic vaginitis or ectopic uterine tissues in endometriosis causes dyspareunia.
Pathophysiology
Sexual pain disorders can therefore be considered as:[1]
- multifactorial
- multisystemic
- complex :
Multifactorial:biological, psychosexual, relational factors can coexist in a woman complaining of coital pain. Over time, these different factors may act as predisposing, precipitating, or perpetuating sexual pain disorders.
Multisystemic: sexual function involves:
- nervous,
- endocrine,
- vascular,
- immunological systems
- vaginal ecosystem
complex: coital pain is greater than the simple peripheral tissue damage that may initially trigger the nociceptive component.When It becomes chronic,the pathophysiology of pain may gradually shift from nociceptive, a friend signal that should induce self protection and defense, to neuropathic, with a progressive involvement of the CNS.
References
- ↑ 10.1007/978-3-319-52539-6_20