Dyspareunia pathophysiology: Difference between revisions
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==Pathophysiology== | ==Pathophysiology== | ||
Sexual pain disorders can therefore be considered as:<ref> 10.1007/978-3-319-52539-6_20</ref><ref name="pmid24811097">{{cite journal |vauthors=Graziottin A, Skaper SD, Fusco M |title=Mast cells in chronic inflammation, pelvic pain and depression in women |journal=Gynecol. Endocrinol. |volume=30 |issue=7 |pages=472–7 |date=July 2014 |pmid=24811097 |doi=10.3109/09513590.2014.911280 |url=}}</ref> | Sexual pain disorders can therefore be considered as:<ref> 10.1007/978-3-319-52539-6_20</ref><ref name="pmid24811097">{{cite journal |vauthors=Graziottin A, Skaper SD, Fusco M |title=Mast cells in chronic inflammation, pelvic pain and depression in women |journal=Gynecol. Endocrinol. |volume=30 |issue=7 |pages=472–7 |date=July 2014 |pmid=24811097 |doi=10.3109/09513590.2014.911280 |url=}}</ref> | ||
*Multifactorial | *'''Multifactorial''' | ||
* | *'''Multisystemic''' | ||
* | *'''Complex''' | ||
'''Multifactorial''':biological, psychosexual, relational factors can coexist in a woman complaining of coital pain. Over time, these different factors may act as predisposing, precipitating, or perpetuating sexual pain disorders.<br> | '''Multifactorial''':biological, psychosexual, relational factors can coexist in a woman complaining of coital pain. Over time, these different factors may act as predisposing, precipitating, or perpetuating sexual pain disorders.<br> | ||
'''Multisystemic''': sexual function involves: | '''Multisystemic''': sexual function involves: | ||
* | *[[Nervous system]] | ||
* | *[[Endocrine system]] | ||
* | *[[Vascular system]] | ||
* | *[[Immunological system|Immunological systems]] | ||
*vaginal ecosystem | *vaginal ecosystem | ||
'''complex''': coital pain is greater than the simple peripheral tissue damage that may initially trigger the nociceptive component.When It becomes chronic,the pathophysiology of pain may gradually shift from nociceptive, a friend signal that should induce self protection and defense, to neuropathic, with a progressive involvement of the CNS.<ref> 10.1007/978-3-319-52539-6_20</ref> | '''complex''': coital pain is greater than the simple peripheral tissue damage that may initially trigger the nociceptive component.When It becomes chronic,the pathophysiology of pain may gradually shift from nociceptive, a friend signal that should induce self protection and defense, to neuropathic, with a progressive involvement of the CNS.<ref> 10.1007/978-3-319-52539-6_20</ref> | ||
Revision as of 00:28, 21 September 2020
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Overview
Dyspareunia is caused by medical causes, psychological problems, and pathophysiology can be considered as multifactorial, multisystemic or complex. dyspareunia vary based on the underlying causes . For example, estrogen deficiency causes atrophic vaginitis or ectopic uterine tissues in endometriosis causes dyspareunia.
Pathophysiology
Sexual pain disorders can therefore be considered as:[1][2]
- Multifactorial
- Multisystemic
- Complex
Multifactorial:biological, psychosexual, relational factors can coexist in a woman complaining of coital pain. Over time, these different factors may act as predisposing, precipitating, or perpetuating sexual pain disorders.
Multisystemic: sexual function involves:
- Nervous system
- Endocrine system
- Vascular system
- Immunological systems
- vaginal ecosystem
complex: coital pain is greater than the simple peripheral tissue damage that may initially trigger the nociceptive component.When It becomes chronic,the pathophysiology of pain may gradually shift from nociceptive, a friend signal that should induce self protection and defense, to neuropathic, with a progressive involvement of the CNS.[3]