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__NOTOC__
{{Vertigo}}


{{CMG}}; {{AE}} {{ZMalik}}
{| class="wikitable"
 
!align="center" style="background: #4479BA; color: #FFFFFF | '''Common Cause of Tremor'''
==Overview==
! align="center" style="background: #4479BA; color: #FFFFFF|  '''Differentiating Feature of Tremor'''
Common physical examination findings associated with vertigo include [[nystagus]] [[hearing]] impairment , [[vision]] changes, and [[imbalance]].
! align="center" style="background: #4479BA; color: #FFFFFF| '''Main Feature of Disease'''
 
|-
==Physical Examination==
| [[Essential tremor]] || Postural Tremor - [[Frequency]] 4–12 Hz, Bilateral onset || [[gait]] [[ataxia]], [[vestibulo-cerebellar]] involvement, reduced by [[alcohol]], [[family history]], [[stress]]/[[fatigue]] can increase tremor [[amplitude]], increases with voluntary movements
Physical examination of patients with vertigo is usually remarkable for [[nystagmus]], [[hearing]] impairment, [[nausea]], [[imbalance]], [[vision]] changes.
|-
 
| [[Parkinson’s disease]] || [[Resting Tremor]] - Unilateral onset || [[Bradykinesia]], [[micrographia]], [[stooped posture]], [[ataxia]], [[rigidity]], [[imbalance]], [[depression]], [[apathy]], decreases with voluntary movements
===Appearance of the Patient===
|-
*Patients with vertigo have variable general appearance depending on the underlying [[etiology]].
| [[Physiologic Tremor]] || Postural tremor - High [[frequency]] 8–10 Hz, low [[amplitude]], irregular oscillations || Tremor occurs while maintaining a posture and mostly disappears if [[eyes]] are closed or a load is placed on the [[muscles]]. Subtle [[innate]] tremor normally present in the general [[population]].
*Unsteady [[gait]] ([[cerebellar]] involvement), [[sensitivity]] to motion/light/[[smell]]/noise (vestibular [[migraine]]) could be observed.
|-
 
| Enhanced [[Physiologic]] Tremor ||Increased [[amplitude]] ||[[Physiologic]] tremor enhanced due to [[fatigue]], [[sleep deprivation]], [[drugs]], [[endocrine disorders]], [[caffeine]], [[stress]].
===Vital Signs===
|-
 
| [[Cerebellar]] Tremor || [[Intention tremor]] - Low [[frequency]] <4 Hz || Occurs in [[multiple sclerosis]], [[stroke]], [[brainstem]] [[tumor]], or [[cerebellar]] [[trauma]]. May feature [[ataxia]], [[dysmetria]], [[dysdiadochokinesia]], and [[dysarthria]].
*[[Blood pressure]] should be assessed to rule out [[orthostatic hypotension]].
|-
*[[Hyperthermia]] could be associated with vestibular [[neuronitis]], [[herpes zoster oticus]], [[acute]] [[labyrinthitis]].
| [[Drug]] Induced Tremor || Can enhance rest, action, postural tremors || [[Amiodarone]], [[bronchodilators]], [[lithium]], [[metoclopramide]], [[neuroleptics]], [[theophylline]], [[valproate]]
 
|-
===Skin===
| [[Orthostatic]] Tremor || [[Essential tremor]] variant, high [[frequency]] 14 Hz-18 Hz|| Occurs in the [[legs]] on standing and is relieved by sitting down
* [[Skin]] [[examination]] of [[patients]] with vertigo is usually normal. However, any [[signs]] of [[trauma]] should be assessed.
|-
 
|Holmes tremor ||  Combination of rest, action, and postural tremors, [[Frequency]] 2Hz-5Hz || Mostly due to [[vascular]] [[lesion]] in [[mesencephalic]], [[thalamic]] or both regions.
===HEENT===
|}
* [[HEENT]] [[examination]] of patients with vertigo is very important. Following [[examinations]] should be performed in every [[patient]] presenting with vertigo:
**[[Otoscopic]] Exam: Can identify [[cholesteatoma]], [[herpes zoster oticus]]([[vesicles]] on [[tympanic membrane]]), [[acute]] [[otitis media]].
**Hennebert’s sign, pushing tragus provokes vertigo or [[nystagmus]] on the affected side in patients with [[perilymphatic fistula]].<ref name="RosenbergGizzi2000">{{cite journal|last1=Rosenberg|first1=Michael L.|last2=Gizzi|first2=Martin|title=NEURO-OTOLOGIC HISTORY|journal=Otolaryngologic Clinics of North America|volume=33|issue=3|year=2000|pages=471–482|issn=00306665|doi=10.1016/S0030-6665(05)70221-8}}</ref>
**[[Hearing]]: Weber or Rinne's test is done in the [[clinic]] or at the bedside to determine if it is conductive or [[sensorineural hearing loss]].
**'''HINTS''': '''H'''ead '''I'''mpulse, '''N'''ystagmus, '''T'''est of '''S'''kew (cover/uncover test) to identify if the cause of [[vestibular neuritis]] is central or peripheral.<ref name="TarnutzerBerkowitz2011">{{cite journal|last1=Tarnutzer|first1=A. A.|last2=Berkowitz|first2=A. L.|last3=Robinson|first3=K. A.|last4=Hsieh|first4=Y.-H.|last5=Newman-Toker|first5=D. E.|title=Does my dizzy patient have a stroke? A systematic review of bedside diagnosis in acute vestibular syndrome|journal=Canadian Medical Association Journal|volume=183|issue=9|year=2011|pages=E571–E592|issn=0820-3946|doi=10.1503/cmaj.100174}}</ref>
**Look for evidence of [[trauma]].
**[[Nystagmus]] assessment is an important feature to distinguish peripheral from the central cause of vertigo:<ref name="pmid20849021">{{cite journal| author=Kaski D, Seemungal BM| title=The bedside assessment of vertigo. | journal=Clin Med (Lond) | year= 2010 | volume= 10 | issue= 4 | pages= 402-5 | pmid=20849021 | doi=10.7861/clinmedicine.10-4-402 | pmc=4952176 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=20849021  }} </ref>
***[[Peripheral]]: Horizontal nystagmus with a torsional component, adaptive.
***Central: Could be in any direction horizontal, vertical, or torsional, non- adaptive.
**[[Dix-Hallpike maneuver]] is used to diagnose [[benign paroxysmal positional vertigo]]<ref name="pmid12392120">{{cite journal| author=Hanley K, O' Dowd T| title=Symptoms of vertigo in general practice: a prospective study of diagnosis. | journal=Br J Gen Pract | year= 2002 | volume= 52 | issue= 483 | pages= 809-12 | pmid=12392120 | doi= | pmc=1316083 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=12392120  }} </ref>. [[Dix-Hallpike maneuver]] can also differentiate between central and peripheral, the intensity of induced symptom decreases with repeated [[maneuvers]] in peripheral but less likely to decrease if the lesion is central in origin.<ref name="pmid10219377">{{cite journal| author=Büttner U, Helmchen C, Brandt T| title=Diagnostic criteria for central versus peripheral positioning nystagmus and vertigo: a review. | journal=Acta Otolaryngol | year= 1999 | volume= 119 | issue= 1 | pages= 1-5 | pmid=10219377 | doi=10.1080/00016489950181855 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=10219377  }} </ref>
 
===Neck===
*[[Neck]] [[examination]] of [[patients]] with vertigo is usually normal. However, any [[signs]] of [[trauma]] should be assessed.
 
===Lungs===
* [[Pulmonary]] [[examination]] of [[patients]] with vertigo is usually normal.
 
===Heart===
* [[Cardiovascular]] [[examination]] of the [[patients]] with vertigo should include heart rate and rhythm, [[pulse]], [[blood pressure]], [[carotid]] [[bruit]], [[orthostatic]] [[blood pressure]] measurement.
 
===Abdomen===
* [[Abdominal]] [[examination]] of [[patients]] with vertigo is usually normal.
 
===Back===
* [[Back]] [[examination]] of [[patients]] with vertigo is usually normal.
 
===Genitourinary===
* [[Genitourinary]] [[examination]] of [[patients]] with vertigo is usually normal.
 
===Neuromuscular===
* [[Neuromuscular]] [[examination]] of [[patients]] with vertigo is very important in identifying the underlying [[etiology]]:
*[[Patient]] is usually oriented to persons, place, and time
*[[Cranial nerves]]: strength, [[sensation]], [[reflexes]] should be [[examined]].
*[[Gait]] [[imbalance]] should be assessed by[[Romberg]] test and tandem walking for [[cerebellar]] [[function]].<ref name="pmid20849021">{{cite journal| author=Kaski D, Seemungal BM| title=The bedside assessment of vertigo. | journal=Clin Med (Lond) | year= 2010 | volume= 10 | issue= 4 | pages= 402-5 | pmid=20849021 | doi=10.7861/clinmedicine.10-4-402 | pmc=4952176 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=20849021 }} </ref>
 
===Extremities===
* Extremities examination of patients with [disease name] is usually normal.
OR
*[[Clubbing]]  
*[[Cyanosis]]  
*Pitting/non-pitting [[edema]] of the upper/lower extremities
*Muscle atrophy
*Fasciculations in the upper/lower extremity
 
==References==
{{Reflist|2}}
 
{{WH}}
{{WS}}
[[Category: (name of the system)]]

Revision as of 20:23, 22 February 2021

Common Cause of Tremor Differentiating Feature of Tremor Main Feature of Disease
Essential tremor Postural Tremor - Frequency 4–12 Hz, Bilateral onset gait ataxia, vestibulo-cerebellar involvement, reduced by alcohol, family history, stress/fatigue can increase tremor amplitude, increases with voluntary movements
Parkinson’s disease Resting Tremor - Unilateral onset Bradykinesia, micrographia, stooped posture, ataxia, rigidity, imbalance, depression, apathy, decreases with voluntary movements
Physiologic Tremor Postural tremor - High frequency 8–10 Hz, low amplitude, irregular oscillations Tremor occurs while maintaining a posture and mostly disappears if eyes are closed or a load is placed on the muscles. Subtle innate tremor normally present in the general population.
Enhanced Physiologic Tremor Increased amplitude Physiologic tremor enhanced due to fatigue, sleep deprivation, drugs, endocrine disorders, caffeine, stress.
Cerebellar Tremor Intention tremor - Low frequency <4 Hz Occurs in multiple sclerosis, stroke, brainstem tumor, or cerebellar trauma. May feature ataxia, dysmetria, dysdiadochokinesia, and dysarthria.
Drug Induced Tremor Can enhance rest, action, postural tremors Amiodarone, bronchodilators, lithium, metoclopramide, neuroleptics, theophylline, valproate
Orthostatic Tremor Essential tremor variant, high frequency 14 Hz-18 Hz Occurs in the legs on standing and is relieved by sitting down
Holmes tremor Combination of rest, action, and postural tremors, Frequency 2Hz-5Hz Mostly due to vascular lesion in mesencephalic, thalamic or both regions.