Spontaneous coronary artery dissection risk factors: Difference between revisions
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* '''[[Fibromuscular dysplasia]]''' | * '''[[Fibromuscular dysplasia]]''' | ||
* '''[[Pregnancy|Pregnancy-related]]:''' [[antepartum]], [[postpartum|early post-partum]], [[postpartum|late post-partum]], [[postpartum|very late post-partum]] | * '''[[Pregnancy|Pregnancy-related]]:''' [[antepartum]], [[postpartum|early post-partum]], [[postpartum|late post-partum]], [[postpartum|very late post-partum]] | ||
* '''[[Pregnancy|Recurrent pregnancies]]:''' [[Parity (medicine)|multiparity | * '''[[Pregnancy|Recurrent pregnancies]]:''' [[Parity (medicine)|multiparity ≥ 4 births]] or [[multigravida]] | ||
* '''[[Connective tissue disorder]]:''' [[Marfan syndrome]], [[Loeys-Dietz syndrome]], [[Ehlers-Danlos syndrome|Ehlers-Danlos syndrome type 4]], [[neurofibromatosis type I]], [[cystic medial necrosis]], [[alpha-1 antitrypsin deficiency]], [[polycystic kidney disease]], [[lysyl oxidase|lysyl oxidase deficiency]], [[Alport syndrome]], [[pseudoxanthoma elasticum]] | * '''[[Connective tissue disorder]]:''' [[Marfan syndrome]], [[Loeys-Dietz syndrome]], [[Ehlers-Danlos syndrome|Ehlers-Danlos syndrome type 4]], [[neurofibromatosis type I]], [[cystic medial necrosis]], [[alpha-1 antitrypsin deficiency]], [[polycystic kidney disease]], [[lysyl oxidase|lysyl oxidase deficiency]], [[Alport syndrome]], [[pseudoxanthoma elasticum]] | ||
* '''[[Systemic inflammation|Systemic inflammatory disease]]:''' [[systemic lupus erythematosus]], [[Crohn's disease]], [[ulcerative colitis]], [[polyarteritis nodosa]], [[sarcoidosis]], [[Churg-Strauss syndrome]], [[Wegener's granulomatosis]], [[rheumatoid arthritis]], [[Kawasaki disease]], [[giant cell arteritis]], [[celiac disease]], [[Takayasu arteritis]], [[cryoglobulinemia]], [[Behçet's disease]] | * '''[[Systemic inflammation|Systemic inflammatory disease]]:''' [[systemic lupus erythematosus]], [[Crohn's disease]], [[ulcerative colitis]], [[polyarteritis nodosa]], [[sarcoidosis]], [[Churg-Strauss syndrome]], [[Wegener's granulomatosis]], [[rheumatoid arthritis]], [[Kawasaki disease]], [[giant cell arteritis]], [[celiac disease]], [[Takayasu arteritis]], [[cryoglobulinemia]], [[Behçet's disease]] | ||
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| style="background: #F5F5F5; padding: 5px;" colspan="2;"| | | style="background: #F5F5F5; padding: 5px;" colspan="2;"| | ||
* '''[[exercise|Intense exercises]]:''' [[isometric exercise]], [[aerobic exercise]] | * '''[[exercise|Intense exercises]]:''' [[isometric exercise]], [[aerobic exercise]] | ||
* ''' | * ''' [[Intense emotional stress|emotional stress]]''' | ||
* '''[[Labor|Labor and delivery]]''' | * '''[[Labor|Labor and delivery]]''' | ||
* '''[[Valsalva maneuver|Valsava-type activities]]:''' [[retching]], [[vomiting]], [[bowel movement]], [[coughing]] | * '''[[Valsalva maneuver|Valsava-type activities]]:''' [[retching]], [[vomiting]], [[bowel movement]], [[coughing]] | ||
Revision as of 16:23, 7 March 2021
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Spontaneous Coronary Artery Dissection Microchapters |
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Differentiating Spontaneous coronary artery dissection from other Diseases |
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Diagnosis |
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Treatment |
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Case Studies |
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Type 1 Type 2A Type 2B Type 3 |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Nate Michalak, B.A. Arzu Kalayci, M.D. [2]
Synonyms and keywords: SCAD
Overview
The risk factors for spontaneous coronary artery dissection include predisposing factors ( vasculopathy, pregnancy, connective tissue disorder, systemic inflammation) and precipitating stressors (e.g., strenuous exercise, emotional stress, recreational drugs).Features that raise the index of suspicion for SCAD include myocardial infarction in young women (age ≤50), absence of traditional cardiovascular risk factors, little or no evidence of coronary atherosclerosis, peripartum state, history of fibromuscular dysplasia, history of connective tissue disorder or systemic inflammation.
Risk Factors
- The phenotypic manifestation of spontaneous coronary artery dissection (SCAD) may occur as a result of predisposing factors compounded by precipitating stressors.[1] *The presence of either predisposing or precipitating factors increases the risk of developing a dissection.
- The potential risk factors for SCAD include:[2][3]
References
- ↑ Saw, Jacqueline; Mancini, G.B. John; Humphries, Karin H. (2016). "Contemporary Review on Spontaneous Coronary Artery Dissection". Journal of the American College of Cardiology. 68 (3): 297–312. doi:10.1016/j.jacc.2016.05.034. ISSN 0735-1097.
- ↑ Saw, J.; Aymong, E.; Sedlak, T.; Buller, C. E.; Starovoytov, A.; Ricci, D.; Robinson, S.; Vuurmans, T.; Gao, M.; Humphries, K.; Mancini, G. B. J. (2014). "Spontaneous Coronary Artery Dissection: Association With Predisposing Arteriopathies and Precipitating Stressors and Cardiovascular Outcomes". Circulation: Cardiovascular Interventions. 7 (5): 645–655. doi:10.1161/CIRCINTERVENTIONS.114.001760. ISSN 1941-7640.
- ↑ Adlam, David; Alfonso, Fernando; Maas, Angela; Vrints, Christiaan; al-Hussaini, Abtehale; Bueno, Hector; Capranzano, Piera; Gevaert, Sofie; Hoole, Stephen P; Johnson, Tom; Lettieri, Corrado; Maeder, Micha T; Motreff, Pascal; Ong, Peter; Persu, Alexandre; Rickli, Hans; Schiele, Francois; Sheppard, Mary N; Swahn, Eva (2018). "European Society of Cardiology, acute cardiovascular care association, SCAD study group: a position paper on spontaneous coronary artery dissection". European Heart Journal. doi:10.1093/eurheartj/ehy080. ISSN 0195-668X.