Dihydrotachysterol: Difference between revisions
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'''Dihydrotachysterol''' (DHT) is a synthetic [[vitamin D]] analog activated in the [[liver]] that does not require [[renal]] hydroxylation like vitamin D2 ([[ergocalciferol]]) and vitamin D3 ([[cholecalciferol]]). DHT has a rapid onset of action (2 hours), a shorter half-life, and a greater effect on [[mineralization]] of bone salts than does vitamin D. | '''Dihydrotachysterol''' (DHT) is a synthetic [[vitamin D]] analog activated in the [[liver]] that does not require [[renal]] hydroxylation like vitamin D2 ([[ergocalciferol]]) and vitamin D3 ([[cholecalciferol]]). DHT has a rapid onset of action (2 hours), a shorter half-life, and a greater effect on [[mineralization]] of bone salts than does vitamin D. | ||
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[[Category:Vitamins]] | [[Category:Vitamins]] | ||
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{{WS}} | {{WS}} |
Revision as of 00:46, 9 August 2012
File:Dihydrotachysterol.svg | |
Clinical data | |
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ATC code | |
Pharmacokinetic data | |
Protein binding | >99% |
Identifiers | |
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CAS Number | |
PubChem CID | |
DrugBank | |
E number | {{#property:P628}} |
ECHA InfoCard | {{#property:P2566}}Lua error in Module:EditAtWikidata at line 36: attempt to index field 'wikibase' (a nil value). |
Chemical and physical data | |
Formula | C28H46O |
Molar mass | 398.664 g/mol |
Dihydrotachysterol (DHT) is a synthetic vitamin D analog activated in the liver that does not require renal hydroxylation like vitamin D2 (ergocalciferol) and vitamin D3 (cholecalciferol). DHT has a rapid onset of action (2 hours), a shorter half-life, and a greater effect on mineralization of bone salts than does vitamin D.
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