Hyperchloremic acidosis: Difference between revisions
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'''Hyperchloremic acidosis''' is a form of [[metabolic acidosis]] associated with a normal [[anion gap]], a decrease in plasma [[bicarbonate]] concentration, and in an increase in plasma [[chloride]] concentration (see [[anion gap]] for a fuller explanation). | '''Hyperchloremic acidosis''' is a form of [[metabolic acidosis]] associated with a normal [[anion gap]], a decrease in plasma [[bicarbonate]] concentration, and in an increase in plasma [[chloride]] concentration (see [[anion gap]] for a fuller explanation). | ||
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* [http://kidney.niddk.nih.gov/kudiseases/pubs/tubularacidosis/ NIH - Renal Tubular Acidosis] | * [http://kidney.niddk.nih.gov/kudiseases/pubs/tubularacidosis/ NIH - Renal Tubular Acidosis] | ||
[[Category:Electrolyte disturbance]] | [[Category:Electrolyte disturbance]] | ||
[[Category:Nephrology]] | [[Category:Nephrology]] | ||
Revision as of 16:08, 9 August 2012
Template:DiseaseDisorder infobox
Hyperchloremic acidosis is a form of metabolic acidosis associated with a normal anion gap, a decrease in plasma bicarbonate concentration, and in an increase in plasma chloride concentration (see anion gap for a fuller explanation).
Causes
- Renal tubular acidosis failure of HCO3- resorption(i.e. proximal renal tubular acidosis) or failure of H+ secretion (ie in distal renal tubular acidosis)
- Renal failure
- Gastrointestinal loss of HCO3- with diarrhea (vomiting will tend to cause hypochloraemic alkalosis).
- Ingestions
- Alcohol (such as ethanol) can effect anion gap by inducing alcohol dehydrogenase enzyme.