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Revision as of 17:26, 9 August 2012


Miglitol
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Clinical data
[[Regulation of therapeutic goods |Template:Engvar data]]
Pregnancy
category
  • AU: B3
  • US: B (No risk in non-human studies)
Routes of
administration
Oral
ATC code
Legal status
Legal status
Pharmacokinetic data
BioavailabilityDose-dependent
Protein bindingNegligible (<4.0%)
MetabolismNil
Elimination half-life2 hours
ExcretionRenal (95%)
Identifiers
CAS Number
PubChem CID
DrugBank
E number{{#property:P628}}
ECHA InfoCard{{#property:P2566}}Lua error in Module:EditAtWikidata at line 36: attempt to index field 'wikibase' (a nil value).
Chemical and physical data
FormulaC8H17NO5
Molar mass207.224 g/mol

WikiDoc Resources for Miglitol

Articles

Most recent articles on Miglitol

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Articles on Miglitol in N Eng J Med, Lancet, BMJ

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Evidence Based Medicine

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Definitions

Definitions of Miglitol

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Directions to Hospitals Treating Miglitol

Risk calculators and risk factors for Miglitol

Healthcare Provider Resources

Symptoms of Miglitol

Causes & Risk Factors for Miglitol

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Treatment of Miglitol

Continuing Medical Education (CME)

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International

Miglitol en Espanol

Miglitol en Francais

Business

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Experimental / Informatics

List of terms related to Miglitol

For patient information, click here

Miglitol is an oral anti-diabetic drug that acts by inhibiting the ability of the patient to breakdown complex carbohydrates into glucose. It is primarily used in diabetes mellitus type 2 for establishing greater glycemic control by preventing the digestion of carbohydrates (such as disaccharides, oligosaccharides, and polysaccharides) into monosaccharides which can be absorbed by the body.

Miglitol inhibits glycoside hydrolase enzymes called alpha-glucosidases. Since miglitol works by preventing digestion of carbohydrates, it lowers the degree of postprandial hyperglycemia. It must be taken at the start of main meals to have maximal effect. Its effect will depend on the amount of non-monosaccharide carbohydrates in a person's diet.

In contrast to acarbose (another alpha-glucosidase inhibitor), miglitol is systemically absorbed; however, it is not metabolized and is excreted by the kidneys.

See also

Template:Oral hypoglycemics


Template:WikiDoc Sources