Peritonitis pathophysiology: Difference between revisions
No edit summary |
|||
Line 1: | Line 1: | ||
__NOTOC__ | |||
{{Peritonitis}} | {{Peritonitis}} | ||
Please help WikiDoc by adding more content here. It's easy! Click [[Help:How_to_Edit_a_Page|here]] to learn about editing. | |||
{{CMG}} | {{CMG}} | ||
==Overview== | ==Overview== | ||
Line 8: | Line 10: | ||
{{Gastroenterology}} | {{Gastroenterology}} | ||
[[Category:Needs content]] | |||
[[Category:Gastroenterology]] | [[Category:Gastroenterology]] | ||
[[Category:Inflammations]] | [[Category:Inflammations]] |
Revision as of 15:54, 4 September 2012
Peritonitis Main Page |
Please help WikiDoc by adding more content here. It's easy! Click here to learn about editing. Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
The peritoneum normally appears greyish and glistening; it becomes dull 2-4 hours after the onset of peritonitis, initially with scarce serous or slightly turbid fluid. Later on, the exudate becomes creamy and evidently suppurative; in dehydrated patients, it also becomes very inspissated. The quantity of accumulated exudate varies widely. It may be spread to the whole peritoneum, or be walled off by the omentum and viscera. Inflammation features infiltration by neutrophils with fibrino-purulent exudation.
References
da:Peritonitis de:Peritonitis it:Peritonite nl:Buikvliesontsteking fi:Peritoniitti sv:Bukhinneinflammation