Progressive multifocal leukoencephalopathy pathophysiology: Difference between revisions
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==Overview== | ==Overview== | ||
PML is a [[demyelinating disease]], in which the [[myelin]] sheath covering the [[axon]]s of nerve cells is gradually destroyed, impairing the transmission of nerve impulses. It affects the white matter, which is mostly composed of axons from the outermost parts of the brain ([[Cerebral cortex|cortex]]). Symptoms include weakness or paralysis, vision loss, impaired speech, and cognitive deterioration. PML is similar to another demyelinating disease, [[multiple sclerosis]], but since it destroys the cells that produce myelin (unlike MS, in which myelin itself is attacked but can be replaced), it progresses much more quickly. Most patients die within four months of onset. | |||
PML destroys [[oligodendrocyte]]s and produces intranuclear inclusions. | |||
==References== | ==References== | ||
{{reflist|2}} | {{reflist|2}} | ||
Revision as of 23:11, 7 February 2012
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Progressive multifocal leukoencephalopathy Microchapters |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
PML is a demyelinating disease, in which the myelin sheath covering the axons of nerve cells is gradually destroyed, impairing the transmission of nerve impulses. It affects the white matter, which is mostly composed of axons from the outermost parts of the brain (cortex). Symptoms include weakness or paralysis, vision loss, impaired speech, and cognitive deterioration. PML is similar to another demyelinating disease, multiple sclerosis, but since it destroys the cells that produce myelin (unlike MS, in which myelin itself is attacked but can be replaced), it progresses much more quickly. Most patients die within four months of onset. PML destroys oligodendrocytes and produces intranuclear inclusions.
References
Template:Gastroenterology Template:Viral diseases de:Progressive multifokale Leukenzephalopathie nl:Progressieve multifocale leukoencefalopathie