Mental disorder risk factors: Difference between revisions
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==Overview== | |||
Risk factors for(Mental disorder)include and | |||
*Prenatal etiologies (e.g., sequence | |||
variations or copy number variants involving one or more genes; chromosomal | |||
disorders), inborn errors of metabolism, brain malformations, maternal disease (including | |||
placental disease), and environmental influences (e.g., alcohol, other drugs, toxins, teratogens). | |||
Perinatal causes include a variety of labor and delivery-related events leading to | |||
neonatal encephalopathy. Postnatal causes include hypoxic ischemic injury, traumatic | |||
brain injury, infections, demyelinating disorders, seizure disorders (e.g., infantile spasms), | |||
severe and chronic social deprivation, and toxic metabolic syndromes and intoxications | |||
(e.g., lead, mercury | |||
==References== | ==References== |
Revision as of 16:28, 10 October 2014
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Kiran Singh, M.D. [2]
Overview
Risk factors for(Mental disorder)include and
- Prenatal etiologies (e.g., sequence
variations or copy number variants involving one or more genes; chromosomal disorders), inborn errors of metabolism, brain malformations, maternal disease (including placental disease), and environmental influences (e.g., alcohol, other drugs, toxins, teratogens). Perinatal causes include a variety of labor and delivery-related events leading to neonatal encephalopathy. Postnatal causes include hypoxic ischemic injury, traumatic brain injury, infections, demyelinating disorders, seizure disorders (e.g., infantile spasms), severe and chronic social deprivation, and toxic metabolic syndromes and intoxications (e.g., lead, mercury