Amusia: Difference between revisions
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Recent research has suggested that the [[human brain]] has [[neural networks]] specifically for the processing of music, and suggests that amusia is caused primarily by a deficiency in fine-grained perception of [[pitch]].<ref>http://www.brams.umontreal.ca/plab/downloads/PeretzHyde03.pdf I. Peretz; K.L. Hyde, ''What is specific to music processing? Insights from congenital amusia'', Trends in Cognitive Sciences, Vol. 7, No. 8, Aug (2003)</ref> | Recent research has suggested that the [[human brain]] has [[neural networks]] specifically for the processing of music, and suggests that amusia is caused primarily by a deficiency in fine-grained perception of [[pitch]].<ref>http://www.brams.umontreal.ca/plab/downloads/PeretzHyde03.pdf I. Peretz; K.L. Hyde, ''What is specific to music processing? Insights from congenital amusia'', Trends in Cognitive Sciences, Vol. 7, No. 8, Aug (2003)</ref> | ||
==References== | ==References== | ||
Revision as of 16:53, 26 November 2012
- For other meanings of Amusia, see Amusia (disambiguation).
Amusia refers to a number of disorders which are indicated by the inability to recognize musical tones or rhythms or to reproduce them. Amusia can be congenital (present at birth) or be acquired sometime later in life (as from brain damage). The term "amusia" is composed of a- + -musia which means the lack of music.
Recent research has suggested that the human brain has neural networks specifically for the processing of music, and suggests that amusia is caused primarily by a deficiency in fine-grained perception of pitch.[1]
References
- ↑ http://www.brams.umontreal.ca/plab/downloads/PeretzHyde03.pdf I. Peretz; K.L. Hyde, What is specific to music processing? Insights from congenital amusia, Trends in Cognitive Sciences, Vol. 7, No. 8, Aug (2003)
- Pearce JM (2005). "Selected observations on amusia". Eur. Neurol. 54 (3): 145–8. doi:10.1159/000089606. PMID 16282692.
- Karl Kleist, Sensory Aphasia and amusia : The myeloarchitectonic basis, Pergamon Press, Oxford, 1962.