21-hydroxylase deficiency secondary prevention: Difference between revisions
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Revision as of 16:23, 20 September 2012
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Congenital adrenal hyperplasia due to 21-hydroxylase deficiency Microchapters |
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Differentiating Congenital adrenal hyperplasia due to 21-hydroxylase deficiency from other Diseases |
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Diagnosis |
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21-hydroxylase deficiency secondary prevention On the Web |
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American Roentgen Ray Society Images of 21-hydroxylase deficiency secondary prevention |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Secondary Prevention
Preventing hyperandrogenism and optimizing fertility
As growth ends, management in girls with CAH changes focus to optimizing reproductive function. Both excessive testosterone from the adrenals and excessive glucocorticoid treatment can disrupt ovulation, resulting in irregularity of menses or amenorrhea, as well as infertility. Continued monitoring of hormone balance and careful readjustment of glucocorticoid dose can usually restore fertility, but as a group, women with CAH have a lower fertility rate than a comparable population.
CAH has little effect on male fertility unless an adult stops taking his glucocorticoid medication entirely for an extended period of time, in which case excessive adrenal testosterone may reduce testicular production as well as spermatogenesis.
References