Ketoacidosis pathophysiology: Difference between revisions
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Created page with "__NOTOC__ {{Ketoacidosis}} {{CMG}} ==Overview== ==References== {{reflist|2}} {{WH}} {{WS}} Category:Medical emergencies Category:Endocrinology Category:Diabetes..." |
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==Overview== | ==Overview== | ||
==Pathophysiology== | |||
Ketoacidosis occurs when cells do not have sufficient glucose to meet their metabolic demands. Instead, [[ketone bodies]] are produced for energy via the [[metabolism]] of [[fatty acids]]. Acidity results from the dissociation of the H<sup>+</sup> ion at physiological pH of metabolic ketone bodies such as [[acetoacetate]], and [[Beta-hydroxybutyrate|β-hydroxybutyrate]]. | |||
==References== | ==References== | ||
{{reflist|2}} | {{reflist|2}} | ||
Revision as of 01:26, 28 September 2012
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Ketoacidosis Microchapters |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Pathophysiology
Ketoacidosis occurs when cells do not have sufficient glucose to meet their metabolic demands. Instead, ketone bodies are produced for energy via the metabolism of fatty acids. Acidity results from the dissociation of the H+ ion at physiological pH of metabolic ketone bodies such as acetoacetate, and β-hydroxybutyrate.