Tricuspid atresia pathophysiology: Difference between revisions
No edit summary |
|||
Line 7: | Line 7: | ||
==Pathophysiology== | ==Pathophysiology== | ||
===Normal Human Heart=== | ===Normal Human Heart=== | ||
[[Image:Diagram of the human heart (cropped).svg|center|Caption:Anterior (frontal) view of the opened heart. White arrows indicate normal blood flow. (Tricuspid valve labeled at bottom left.)]] | [[Image:Diagram of the human heart (cropped).svg|center|500px|Caption:Anterior (frontal) view of the opened heart. White arrows indicate normal blood flow. (Tricuspid valve labeled at bottom left.)]] | ||
===Tricuspid Atresia=== | ===Tricuspid Atresia=== |
Revision as of 19:26, 2 October 2012
Tricuspid atresia Microchapters |
Diagnosis |
---|
Treatment |
Special Scenarios |
Case Studies |
Tricuspid atresia pathophysiology On the Web |
American Roentgen Ray Society Images of Tricuspid atresia pathophysiology |
Risk calculators and risk factors for Tricuspid atresia pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor-In-Chief: Keri Shafer, M.D. [2] Priyamvada Singh, MBBS [[3]]; Assistant Editor-In-Chief: Kristin Feeney, B.S. [[4]]
Overview
Pathophysiology
Normal Human Heart
Tricuspid Atresia
This defect occurs during prenatal development. Because of the lack of an A-V connection, an atrial septal defect (ASD) must be present to maintain blood flow. Also, since there is a lack of a right ventricle there must be a way to pump blood into the pulmonary arteries, and this is accomplished by a ventricular septal defect (VSD).
Blood is mixed in the left atrium. Because the only way the pulmonary circulation receives blood is through the VSD, a patent ductus arteriosus is usually also formed to increase pulmonary flow.