Cyclosporiasis pathophysiology: Difference between revisions
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Revision as of 21:19, 21 November 2012
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Cyclosporiasis Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Cyclosporiasis pathophysiology On the Web |
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American Roentgen Ray Society Images of Cyclosporiasis pathophysiology |
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Risk calculators and risk factors for Cyclosporiasis pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Pathophysiology
Some of elements of this figure were created based on an illustration by Ortega et al. Cyclospora cayetanensis. In: Advances in Parasitology: opportunistic protozoa in humans. San Diego: Academic Press; 1998. p. 399-418.
When freshly passed in stools, the oocyst is not infective (thus, direct fecal-oral transmission cannot occur; this differentiates Cyclospora from another important coccidian parasite, Cryptosporidium). In the environment , sporulation occurs after days or weeks at temperatures between 22°C to 32°C, resulting in division of the sporont into two sporocysts, each containing two elongate sporozoites . Fresh produce and water can serve as vehicles for transmission and the sporulated oocysts are ingested (in contaminated food or water) . The oocysts [[excyst] in the gastrointestinal tract, freeing the sporozoites which invade the epithelial cells of the small intestine . Inside the cells they undergo asexual multiplication and sexual development to mature into oocysts, which will be shed in stools . The potential mechanisms of contamination of food and water are still under investigation.