Atrioventricular block pathophysiology: Difference between revisions

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Revision as of 16:04, 5 February 2013

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Pathophysiology

Atrioventricular (AV) block is caused by one of the following mechanisms i.e. fibrosis or degeneration of the conduction system, ischemic heart disease, or medications.

References

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 ==Pathophysiology==
-Atrioventricular (AV) block is caused by one of the following mechanisms i.e. [[fibrosis]] or degeneration of the conduction system, [[ischemic heart disease]], or medications. AV block is categorized by degree and site of conduction block. In first-degree AV block, all atrial impulses are conducted to the ventricle. However, there is a delay within the [[AV node]], resulting in a [[prolonged PR interval]] on [[ECG]] (>200 msec or >5 small blocks). [[Second-degree AV block]] can be of one of the two types: [[Mobitz type I]] and [[Mobitz type II]]. Mobitz I, or [[Wenckebach block]] , consists of progressive prolongation of PR interval, until loss of conduction to the ventricle occurs (missed beat). Mobitz I block is rarely symptomatic and does not require treatment. On the other hand, Mobitz II AV block is characterized by a constant PR interval with intermittent missed beats. The missed beats can occur with varying frequency such as occasional to 3:1 or 2:1. [[Complete heart block]] (third-degree heart block) is characterized by a complete lack of conduction from the atria to the ventricles. The ventricular rates in complete heart blocks are slower than the atrial rate. A junctional escape rate is generally between 40 and 60 beats/min and shows narrow QRS complex on EKG, whereas a ventricular escape rate is slower with a wide QRS complex.
+Atrioventricular (AV) block is caused by one of the following mechanisms i.e. [[fibrosis]] or degeneration of the conduction system, [[ischemic heart disease]], or medications.
 ==References==
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Atrioventricular block pathophysiology: Difference between revisions

Revision as of 16:04, 5 February 2013

Pathophysiology

References

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