Presbycusis causes: Difference between revisions
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==References== | ==References== | ||
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[[Category: | [[Category:Primary care]] | ||
[[Category:Disease]] | [[Category:Disease]] | ||
[[Category: | [[Category:Hearing]] | ||
[[Category:Needs content]] | [[Category:Needs content]] | ||
[[Category:Needs overview]] | [[Category:Needs overview]] | ||
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{{WikiDoc Sources}} | {{WikiDoc Sources}} |
Revision as of 15:59, 26 April 2013
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Saumya Easaw, M.B.B.S.[2]
Causes
Common Causes
- Hereditary: Features like early aging of the cochlea and susceptibility of the cochlea for drug insults are genetically determined.
- Atherosclerosis: May diminish vascularity of the cochlea, thereby reducing its oxygen supply.
- Dietary habits: Increased intake of fatty diet may accelerate atherosclerotic changes in old age.
- Diabetes: May cause vasculitis and endothelial proliferation in the blood vessels of the cochlea, thereby reducing its blood supply.
- Noise trauma: Exposure to loud noise on a continuing basis stresses the already hypoxic cochlea, hastening the presbycusis process.
- Smoking: Is postulated to accentuate atherosclerotic changes in blood vessels aggravating presbycusis.
- Hypertension: Causes potent vascular changes, like reduction in blood supply to the cochlea, thereby aggravating presbycusis.
- Ototoxic drugs: Ingestion of ototoxic drugs like aspirin may hasten the process of presbycusis.