Cardiac amyloidosis electrocardiogram: Difference between revisions
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* Left ventricular hypertrophy pattern | * Left ventricular hypertrophy pattern | ||
* Higher degree AV blocks | * Higher degree AV blocks | ||
* [[Bundle branch block]]s | |||
* AV nodal slowing | * AV nodal slowing | ||
*[[Arrhythmia]]s such as | * [[Arrhythmia]]s such as | ||
** [[Atrial fibrillation]] | ** [[Atrial fibrillation]] | ||
** [[Atrial flutter]] | ** [[Atrial flutter]] | ||
Revision as of 04:01, 5 May 2013
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Cardiac amyloidosis Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Cardiac amyloidosis electrocardiogram On the Web |
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American Roentgen Ray Society Images of Cardiac amyloidosis electrocardiogram |
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Risk calculators and risk factors for Cardiac amyloidosis electrocardiogram |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Raviteja Guddeti, M.B.B.S. [2]; Aarti Narayan, M.B.B.S [3]; Cafer Zorkun, M.D., Ph.D. [4]
Overview
The combination of low voltage electrocardiographic pattern and increased thickness of the left ventricular posterior wall and interventricular septum on echocardiogram is highly specific for cardiac amyloidosis. Conduction and rhythm disturbances are common in cardiac amyloidosis, however direct infiltration of the specialized conduction tissue of the heart by the amyloid does not account for the majority of these disturbances.
Electrocardiogram
Electrocardiographic findings in cardiac amyloidosis incluse:[1][2][3]
- Low voltage complexes
- Pseudo-infarct pattern (poor R wave progression)
- Left ventricular hypertrophy pattern
- Higher degree AV blocks
- Bundle branch blocks
- AV nodal slowing
- Arrhythmias such as
Lack of variability in heart rate due to autonomic dysfunction can be found on Holter monitoring and also it is an important predictor of 1-year mortality.[4] This lack of variability in heart rate was shown to be a consistent predictive parameter of short-term mortality in patients with AL type cardiac amyloidosis compared to those with AA type.
References
- ↑ Ridolfi RL, Bulkley BH, Hutchins GM (1977). "The conduction system in cardiac amyloidosis. Clinical and pathologic features of 23 patients". The American Journal of Medicine. 62 (5): 677–86. PMID 871125. Unknown parameter
|month=ignored (help) - ↑ Rahman JE, Helou EF, Gelzer-Bell R; et al. (2004). "Noninvasive diagnosis of biopsy-proven cardiac amyloidosis". Journal of the American College of Cardiology. 43 (3): 410–5. doi:10.1016/j.jacc.2003.08.043. PMID 15013123. Unknown parameter
|month=ignored (help) - ↑ Murtagh B, Hammill SC, Gertz MA, Kyle RA, Tajik AJ, Grogan M (2005). "Electrocardiographic findings in primary systemic amyloidosis and biopsy-proven cardiac involvement". The American Journal of Cardiology. 95 (4): 535–7. doi:10.1016/j.amjcard.2004.10.028. PMID 15695149. Unknown parameter
|month=ignored (help) - ↑ Reyners AK, Hazenberg BP, Reitsma WD, Smit AJ (2002). "Heart rate variability as a predictor of mortality in patients with AA and AL amyloidosis". European Heart Journal. 23 (2): 157–61. doi:10.1053/euhj.2001.2972. PMID 11785998. Unknown parameter
|month=ignored (help)