Wide complex tachycardia resident survival guide

Wide Complex Tachycardia Resident Survival Guide Microchapters
Overview
Causes
FIRE
Diagnosis
Treatment
Do's
Don'ts

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Rim Halaby, M.D. [2]

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Overview

Wide complex tachycardia is characterized by a heart rate more than 100 beats per minute associated with a QRS interval of more than 120 ms. When wide complex tachycardia is present, it is important to determine whether the tachycardia is of a supraventricular or a ventricular origin.

Causes

Life Threatening Causes

Life-threatening causes include conditions which may result in death or permanent disability within 24 hours if left untreated. Wide complex tachycardia may be a life-threatening condition and must be treated as such irrespective of the causes.

Common Causes

• Atrial fibrillation with aberrancy
• Atrial fibrillation with pre-excitation
• Supraventricular tachycardia (SVT) with aberrancy
• Ventricular tachycardia

Click here for the complete list of causes.

FIRE: Focused Initial Rapid Evaluation

A Focused Initial Rapid Evaluation (FIRE) as shown below should be performed to identify patients in need of immediate intervention.

Boxes in red signify that an urgent management is needed.

Complete Diagnostic Approach

Shown below is an algorithm depicting the complete diagnostic approach to wide complex tachycardia according to the 2003 ACC/AHA/ESC guidelines for the management of patients with supraventricular arrhythmias and the 2005 American Heart Association guidelines for cardiopulmonary resuscitation and emergency cardiovascular care.

Click on boxes to expand/collapse detailed information. Characterize the symptoms                                                   Characterize the timing of the symptoms                                                   Identify possible triggers                                                   ❑ Examine the patient ❑ Order an EKG

Characterize the symptoms ❑ Asymptomatic ❑ Palpitations ❑ Dyspnea ❑ Fatigue ❑ Chest discomfort ❑ Lightheadedness ❑ Syncope Characterize the timing of the symptoms ❑ Onset ❑ Duration ❑ Frequency Identify possible triggers ❑ Infection ❑ Caffeine ❑ Alcohol ❑ Nicotine ❑ Recreational drugs ❑ Hypovolemia ❑ Hyperthyroidism ❑ Hypoxia ❑ Acidosis ❑ Hypokalemia ❑ Hyperkalemia ❑ Hypoglycemia ❑ Hypothermia ❑ Toxins ❑ Cardiac tamponade ❑ Pulmonary embolism ❑ Coronary thrombosis ❑ Trauma

Treatment

Shown below is an algorithm depicting the therapeutic approach of wide complex tachycardia according to the 2003 ACC/AHA/ESC guidelines for the management of patients with supraventricular arrhythmias and the 2005 American Heart Association guidelines for cardiopulmonary resuscitation and emergency cardiovascular care.

ABC: Air, breathing and circulation; Afib: Atrial fibrillation; BBB: Bundle branch block; LV: Left ventricle; SVT: Supraventricular tachycardia; VT: Ventricular tachycardia; WPW: Wold Parkinson White

Click on boxes to expand/collapse detailed information.                     Wide complex tachycardia QRS ≥ 120ms                                                                                             Begin initial management                                                                                             Determine if the patient has any unstable sign or symptom                                                                                                                 No               Yes                                                                         Determine the regularity of the rhythm               Perform immediate synchronized cardioversion                                                                           Regular rhythm               Irregular rhythm                                                                                                           VT or uncertain rhythm   SVT with aberrancy   Afib with aberrancy   Pre-excited Afib (Afib + WPW)   Recurrent polymorphic VT   Torsade de pointes

Begin initial management ❑ Assess and support ABC's as needed ❑ Give oxygen ❑ Monitor ECG, blood pressure, oxymetry ❑ Establish IV access ❑ Identify and treat reversible causes Determine if the patient has any unstable sign or symptom ❑ Chest pain ❑ Congestive heart failure ❑ Hypotension ❑ Loss of consciousness ❑ Seizures Perform immediate synchronized cardioversion ❑ Perform immediate synchronized cardioversion ❑ Give IV sedation if the patient is conscious ❑ Consider expert consultation VT or uncertain rhythm ❑ Give amiodarone 150 mg IV over 10 min ❑ Repeat amiodarone as needed for a maximal dose of 2.2g/24h ❑ Prepare for elective synchronized cardioversion SVT with aberrancy ❑ Attempt vagal maneuvers ❑ Give adenosine 6 mg rapid IV push ❑ If no conversion give 12 mg IV push ❑ May repeat 12 mg dose once Afib with aberrancy ❑ Consider expert consultation ❑ Control rate e.g diltiazem or beta blockers Pre-excited Afib (Afib + WPW) ❑ Consider expert consultation ❑ Avoid AV nodal blocking agents e.g adenosine, digoxin, diltiazem and verapamil ❑ Consider amiodarone 150 mg IV over 10 min Recurrent polymorphic VT ❑ Consider expert consultation Torsade de pointes ❑ Load with magnesium 1-2 g over 5-60 min, then infusion

Differentiating SVT from VT

Shown below is a table summarizing some clues that help differentiate SVT from VT.

Clues	Type of arrhythmia
Irregularly irregular rhythm	Atrial fibrillation or atrial flutter with aberrancy
Previous myocardial infarction or structural heart disease	Ventricular tachycardia
Ventricular rate faster than atrial rate	Ventricular tachycardia
Typical RBBB or LBBB	Supraventricular tachycardia
Precordial leads: ❑ Concordant ❑ No R/S pattern ❑ Onset of R to nadir longer than 100ms	Ventricular tachycardia
RBBB pattern: ❑ qR, Rs or Rr' in V1 ❑ Frontal plane axis range from +90 degrees to -90 degrees	Ventricular tachycardia
LBBB pattern: ❑ R in V1 longer than 30 ms ❑ R to nadir of S in V1 greater than 60 ms ❑ qR or qS in V6	Ventricular tachycardia

For more details about differentiating VT from SVT, click here

Antiarrhythmics

Shown below is a table summarizing the choices of the antiarrhythmic drugs for the different types of tachycardia according to the 2003 ACC/AHA/ESC guidelines for the management of patients with supraventricular arrhythmias.

BBB: Bundle branch block; LV: Left ventricle; SVT: Supraventricular tachycardia

Management of wide complex tachycardia Ventricular tachycardia or wide QRS tachycardia of unknown origin ▸ Procainamide (Class I, level of evidence B) OR ▸ Sotalol (Class I, level of evidence B) OR ▸ Amiodarone OR ▸ Cardioversion (Class I, level of evidence B) OR ▸ Lidocaine (Class IIb, level of evidence B) OR ▸ Adenosine (Class IIb, level of evidence C) OR ▸ Beta blocker (Class III, level of evidence C) OR ▸ Verapamil (Class III, level of evidence B) Wide QRS tachycardia of unknown origin + poor LV function ▸ Amiodarone (Class I, level of evidence B) OR ▸ Cardioversion (Class I, level of evidence B) SVT + BBB ▸ Vagal maneuvers (Class I, level of evidence B) OR ▸ Adenosine (Class I, level of evidence A) OR ▸ Verapamil (Class I, level of evidence A) OR ▸ Diltiazem (Class I, level of evidence A) OR ▸ Beta blocker (Class IIb, level of evidence C) OR ▸ Amiodarone (Class IIb, level of evidence C) OR ▸ Digoxin (Class IIb, level of evidence C) SVT or atrial fibrillation + Preexcitation ▸ Flecainide (Class I, level of evidence B) OR ▸ Ibutilide (Class I, level of evidence B) OR ▸ Procainamide (Class I, level of evidence B) OR ▸ Cardioversion (Class I, level of evidence C)

Do's

• Refer the patient to an arrhythmia specialist when the tachycardia causes syncope or dyspnea as well as when the wide complex tachycardia is of unknown cause.
• Place an ambulatory 24 hour Holter when the tachycardia is frequent and transient.
• Treat the patient as having a ventricular tachycardia when the diagnosis of supraventricular tachycardia can not be made.
• Suspect ventricular tachycardia in a patient with wide complex tachycardia and previous myocardial infarction or a history of structural heart disease.
• Use beta blockers with caution in pulmonary diseases or congestive heart failure.

Dont's

• Don't rely on the hemodynamic status of the patient and the heart rate to differenciate SVT from VT.

References