Cardiogenic shock history and symptoms: Difference between revisions
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== History and Symptoms == | == History and Symptoms == | ||
* [[Anxiety]], agitation, restlessness, and an [[Glasgow Coma Scale|altered mental state]] including flacid [[coma]] may be present due to decreased cerebral perfusion and ensuing [[hypoxia (medical)|hypoxia]]. | * [[Anxiety]], agitation, restlessness, and an [[Glasgow Coma Scale|altered mental state]] including flacid [[coma]] may be present due to decreased [[cerebral]] [[perfusion]] and ensuing [[hypoxia (medical)|hypoxia]]. | ||
* [[Fatigue]] may be present due to the work of breathing and [[hypoxia]]. | * [[Fatigue]] may be present due to the work of [[breathing]] and [[hypoxia]]. | ||
==References== | ==References== | ||
Revision as of 16:10, 22 May 2014
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: João André Alves Silva, M.D. [2]
Overview
Attending to the catastrophic outcome of cardiogenic shock in a very short time span, its diagnosis must be reached as early as possible in order for proper therapy to be started. This period until diagnosis and treatment initiation is particularly important in the case of cardiogenic shock since the mortality rate of this condition complicating acute-MI is very high, along with the fact that the ability to revert the damage caused, through reperfusion techniques, declines considerably with diagnostic delays. Therefore and due to the unstable state of these patients, the diagnostic evaluations are usually performed as supportive measures are initiated. The diagnostic measures should start with the proper history and physical examination, including blood pressure beasurements, followed by an EKG, chest x-ray and collection of blood samples for evaluation. The physician should have in mind the common features of shock, irrespective of the type of shock, in order to avoid delays in the diagnosis. Although not all shock patients present in the same way, these features include: abnormal mental status, cool extremities, clammy skin, manifestations of hypoperfusion, such as hypotension, oliguria and evidence of metabolic acidosis on the blood results.[1]
History and Symptoms
- Anxiety, agitation, restlessness, and an altered mental state including flacid coma may be present due to decreased cerebral perfusion and ensuing hypoxia.
- Fatigue may be present due to the work of breathing and hypoxia.
References
- ↑ Longo, Dan L. (Dan Louis) (2012). Harrison's principles of internal medici. New York: McGraw-Hill. ISBN 978-0-07-174889-6.