Cardiogenic shock physical examination: Difference between revisions
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===Vital Signs=== | ===Vital Signs=== | ||
* [[Hypotension]] may be present due to a decrease in [[cardiac output]]. | * [[Hypotension]] may be present due to a decrease in [[cardiac output]]. | ||
* [[Tachycardia]] with a rapid, weak, thready rapid pulse is present. | * [[Tachycardia]] with a rapid, weak, thready rapid [[pulse]] is present. | ||
*[[Pulse pressure]] is reduced. | *[[Pulse pressure]] is reduced. | ||
===Neck=== | ===Neck=== | ||
* Distended [[jugular vein]]s due to increased [[jugular venous pressure]]. | * Distended [[jugular vein]]s due to increased [[jugular venous pressure]]. | ||
===Skin=== | ===Skin=== | ||
* [[Cyanosis]], [[Cool extremities|cool]], [[Cool extremities|clammy]], and mottled skin ([[cutis marmorata]]), due to [[vasoconstriction]] and subsequent [[hypoperfusion]] of the [[skin]] are often present. | * [[Cyanosis]], [[Cool extremities|cool]], [[Cool extremities|clammy]], and mottled skin ([[cutis marmorata]]), due to [[vasoconstriction]] and subsequent [[hypoperfusion]] of the [[skin]] are often present. | ||
===Lungs=== | ===Lungs=== | ||
* Rapid and deep respirations ([[hyperventilation]]) due to [[sympathetic nervous system]] stimulation by [[stretch receptors]] and as compensation for [[metabolic acidosis]]. | * Rapid and deep respirations ([[hyperventilation]]) due to [[sympathetic nervous system]] stimulation by [[stretch receptors]] and as compensation for [[metabolic acidosis]]. | ||
* [[Pulmonary edema]] (fluid in the [[lungs]]) due to insufficient pumping of the [[heart]], leading to fluid accumulation. | |||
* [[Pulmonary edema]] (fluid in the lungs) due to insufficient pumping of the heart, fluid | |||
===Genitourinary=== | ===Genitourinary=== | ||
* [[Oliguria]] ([[low urine output]]) due insufficient [[renal]] [[perfusion]] | * [[Oliguria]] ([[low urine output]]) due insufficient [[renal]] [[perfusion]]. | ||
==References== | ==References== | ||
Revision as of 01:54, 23 May 2014
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Cardiogenic Shock Microchapters |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: João André Alves Silva, M.D. [2]
Overview
Attending to the catastrophic outcome of cardiogenic shock in a very short time span, its diagnosis must be reached as early as possible in order for proper therapy to be started. This period until diagnosis and treatment initiation is particularly important in the case of cardiogenic shock since the mortality rate of this condition complicating acute-MI is very high, along with the fact that the ability to revert the damage caused, through reperfusion techniques, declines considerably with diagnostic delays. Therefore and due to the unstable state of these patients, the diagnostic evaluations are usually performed as supportive measures are initiated. The diagnostic measures should start with the proper history and physical examination, including blood pressure beasurements, followed by an EKG, chest x-ray and collection of blood samples for evaluation. The physician should have in mind the common features of shock, irrespective of the type of shock, in order to avoid delays in the diagnosis. Although not all shock patients present in the same way, these features include: abnormal mental status, cool extremities, clammy skin, manifestations of hypoperfusion, such as hypotension and oliguria, as well as evidence of metabolic acidosis on the blood results.[1]
Physical Examination
Vital Signs
- Hypotension may be present due to a decrease in cardiac output.
- Tachycardia with a rapid, weak, thready rapid pulse is present.
- Pulse pressure is reduced.
Neck
- Distended jugular veins due to increased jugular venous pressure.
Skin
- Cyanosis, cool, clammy, and mottled skin (cutis marmorata), due to vasoconstriction and subsequent hypoperfusion of the skin are often present.
Lungs
- Rapid and deep respirations (hyperventilation) due to sympathetic nervous system stimulation by stretch receptors and as compensation for metabolic acidosis.
- Pulmonary edema (fluid in the lungs) due to insufficient pumping of the heart, leading to fluid accumulation.
Genitourinary
- Oliguria (low urine output) due insufficient renal perfusion.
References
- ↑ Longo, Dan L. (Dan Louis) (2012). Harrison's principles of internal medici. New York: McGraw-Hill. ISBN 978-0-07-174889-6.