Livedoid vasculitis: Difference between revisions
Jump to navigation
Jump to search
Ochuko Ajari (talk | contribs) |
Ochuko Ajari (talk | contribs) |
||
| Line 140: | Line 140: | ||
===Causes in Alphabetical Order=== | ===Causes in Alphabetical Order=== | ||
{{col-begin|width=80%}} | |||
{{col-break|width=33%}} | |||
*[[Amantadine]] | |||
*[[Anticardiolipin syndrome]] | |||
*[[Antiphospholipid syndrome]] | |||
*[[Antithrombin III deficiency]] | |||
*[[Arteriosclerosis]] | |||
*[[Buerger's disease]] | |||
*[[Carbon dioxide arteriography]] | |||
*[[Cholesterol emboli syndrome]] | |||
*[[Churg-Strauss syndrome]] | |||
*[[Cryoglobulinemia]] | |||
*[[Cutis marmorata telangiectatica congenita]] | |||
*[[Cystathionine beta-synthase deficiency]] | |||
*[[Cytomegalovirus infection]] | |||
*[[Dermal vascular occlusion]] | |||
*[[Dermatomyositis]] | |||
*[[Dextroamphetamine]] | |||
*[[Down syndrome]] | |||
*[[Ehlers-Danlos syndrome]] | |||
*[[Elevated fibrinopeptide A]] | |||
*[[Factor V Leiden mutation]] | |||
*[[Familial primary cryofibrinogenemia]] | |||
*[[FILS syndrome]] | |||
*[[Gefitinib]] | |||
*[[Henoch-Schönlein purpura]] | |||
*[[Homocystinuria]] | |||
*[[Hypercalcaemia]] | |||
*[[Hyperhomocysteinemia]] | |||
*[[livedo reticularis|Idiopathic livedo reticularis]] | |||
*[[IgM antiphosphatidylserine-prothrombin complex antibody]] | |||
*[[Immune complex disease]] | |||
*[[Increased plasminogen activator release]] | |||
*[[Interferon type I|Interferon beta treatment]] | |||
*[[Lupus anticoagulant]] | |||
{{col-break|width=33%}} | |||
*[[Lyme disease]] | |||
*[[Lymphoma]] | |||
*[[breast cancer|Metastatic breast carcinoma]] | |||
*[[Methylphenidate]] | |||
*[[catheter|Midline catheter]] | |||
*[[Mixed connective tissue disease]] | |||
*[[Mononeuritis multiplex]] | |||
*[[Moyamoya disease]] | |||
*[[Oxalosis]] | |||
*[[Pancreatitis]] | |||
*[[Peripheral arterial occlusive disease]] | |||
*[[Pernicious anaemia]] | |||
*[[Pheochromocytoma]] | |||
*[[Plasminogen activator inhibitor-1 promoter homozygosity]] | |||
*[[Polyarteritis nodosa]] | |||
*[[Polyclonal IgM hypergammopathy]] | |||
*[[Polycythaemia rubra vera]] | |||
*[[Primary hyperoxaluria]] | |||
*[[Protein C deficiency]] | |||
*[[Prothrombin g20210a heterozygosity]] | |||
*[[Rasagiline]] | |||
*[[Renal cell carcinoma]] | |||
*[[Rheumatoid arthritis]] | |||
*[[Sarcoidosis]] | |||
*[[Scleroderma]] | |||
*[[breast implant|Silicone implants]] | |||
*[[Sjögren's syndrome]] | |||
*[[Sneddon-Champion syndrome]] | |||
*[[Syphilis]] | |||
*[[Systemic lupus erythematosus]] | |||
*[[Thrombocytosis]] | |||
*[[Tuberculosis]] | |||
*[[Vasculitis]] | |||
{{col-end}} | |||
==Treatment== | ==Treatment== | ||
Revision as of 20:34, 29 June 2014
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Synonyms and keywords: Livedo reticularis; livedo vasculitis; livedoid vasculopathy; primary livedo reticularis; idiopathic livedo reticularis; vitiligo reticularis
Overview
Livedoid vasculitis is a vascular disorder mostly affecting women. It may be aggravated by exposure to cold and occurs most often in the lower extremities. It can also be associated with the presence of anti-cardiolipin antibodies (the Antiphospholipid syndrome).
Historical Perspective
The condition's name derives from the Latin livere meaning bluish.
Causes
Life Threatening Causes
Life-threatening causes include conditions which may result in death or permanent disability within 24 hours if left untreated.
Common Causes
- Anticardiolipin antibodies
- Antiphospholipid syndrome
- Factor V Leiden mutation
- Hyperhomocysteinemia
- Lupus anticoagulant
- Protein C deficiency
Causes by Organ System
| Cardiovascular | No underlying causes |
| Chemical/Poisoning | No underlying causes |
| Dental | No underlying causes |
| Dermatologic | No underlying causes |
| Drug Side Effect | No underlying causes |
| Ear Nose Throat | No underlying causes |
| Endocrine | No underlying causes |
| Environmental | No underlying causes |
| Gastroenterologic | No underlying causes |
| Genetic | No underlying causes |
| Hematologic | No underlying causes |
| Iatrogenic | No underlying causes |
| Infectious Disease | No underlying causes |
| Musculoskeletal/Orthopedic | No underlying causes |
| Neurologic | No underlying causes |
| Nutritional/Metabolic | No underlying causes |
| Obstetric/Gynecologic | No underlying causes |
| Oncologic | No underlying causes |
| Ophthalmologic | No underlying causes |
| Overdose/Toxicity | No underlying causes |
| Psychiatric | No underlying causes |
| Pulmonary | No underlying causes |
| Renal/Electrolyte | No underlying causes |
| Rheumatology/Immunology/Allergy | No underlying causes |
| Sexual | No underlying causes |
| Trauma | No underlying causes |
| Urologic | No underlying causes |
| Miscellaneous | No underlying causes |
Causes in Alphabetical Order
Treatment
Other than identifying and treating any underlying conditions in secondary livedo,[1] idiopathic livedo reticularis itself may improve with warming the legs, but once established the skin discolouration may become permanent.
References
- ↑ Fleischer A, Resnick S (1990). "Livedo reticularis". Dermatol Clin. 8 (2): 347–54. PMID 2191805.