Angiomyolipoma pathophysiology: Difference between revisions
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{{Angiomyolipoma}} | {{Angiomyolipoma}} | ||
{{CMG}}''' Associate Editor-In-Chief:''' {{CZ}} | {{CMG}}''' Associate Editor-In-Chief:''' {{CZ}} | ||
==Pathophysiology== | ==Pathophysiology== | ||
Since all three components of an angiomyolipoma (vascular cells, immature smooth muscle cells and fat cells) contain a [[Two-hit hypothesis|"second hit" mutation]], they are believed to have derived from a common [[progenitor cell]] that suffered the common second hit mutation. | Since all three components of an angiomyolipoma (vascular cells, immature smooth muscle cells and fat cells) contain a [[Two-hit hypothesis|"second hit" mutation]], they are believed to have derived from a common [[progenitor cell]] that suffered the common second hit mutation. | ||
Revision as of 17:49, 14 August 2015
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Angiomyolipoma Microchapters |
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Diagnosis |
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Treatment |
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Case Studies |
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Angiomyolipoma pathophysiology On the Web |
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American Roentgen Ray Society Images of Angiomyolipoma pathophysiology |
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Risk calculators and risk factors for Angiomyolipoma pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor-In-Chief: Cafer Zorkun, M.D., Ph.D. [2]
Pathophysiology
Since all three components of an angiomyolipoma (vascular cells, immature smooth muscle cells and fat cells) contain a "second hit" mutation, they are believed to have derived from a common progenitor cell that suffered the common second hit mutation.
Microscopic Pathology
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