Carcinoid syndrome causes: Difference between revisions
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==Causes== | ==Causes== | ||
*Occasionally, GI carcinoids occur in association with inherited syndromes, such as MEN1 and NF1.<ref>{{Cite web | title =Molecular genetics | url = | *Occasionally, GI carcinoids occur in association with inherited syndromes, such as MEN1 and NF1.<ref>{{Cite web | title =Molecular genetics | ||
| url =http://www.cancer.gov/types/gi-carcinoid-tumors/hp/gi-carcinoid-treatment-pdq#section/_21 }}</ref> | |||
*In sporadic GI carcinoids, numerous chromosomal imbalances have been found by comparative genome hybridization analysis. Gains involving chromosomes 5, 14, 17 (especially 17q), and 19 and losses involving chromosomes 11 (especially 11q) and 18 appear to be the most common. | *In sporadic GI carcinoids, numerous chromosomal imbalances have been found by comparative genome hybridization analysis. Gains involving chromosomes 5, 14, 17 (especially 17q), and 19 and losses involving chromosomes 11 (especially 11q) and 18 appear to be the most common. | ||
==References== | ==References== | ||
Revision as of 14:12, 17 September 2015
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]Associate Editor(s)-in-Chief: Parminder Dhingra, M.D. [2]
Overview
Causes
- Occasionally, GI carcinoids occur in association with inherited syndromes, such as MEN1 and NF1.[1]
- In sporadic GI carcinoids, numerous chromosomal imbalances have been found by comparative genome hybridization analysis. Gains involving chromosomes 5, 14, 17 (especially 17q), and 19 and losses involving chromosomes 11 (especially 11q) and 18 appear to be the most common.