Angiomyolipoma differential diagnosis: Difference between revisions
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{{Angiomyolipoma}} | {{Angiomyolipoma}} | ||
{{CMG}} {{AE}}{{Faizan}} | {{CMG}} {{AE}}{{Faizan}} | ||
When an AML has typical appearances there is essentially no differential. If atypical, especially when fat-poor other lesions to consider include: | When an AML has typical appearances there is essentially no differential. If atypical, especially when fat-poor other lesions to consider include: | ||
*retroperitoneal liposarcoma invading the kidney: | *retroperitoneal liposarcoma invading the kidney: | ||
Revision as of 14:55, 21 September 2015
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Faizan Sheraz, M.D. [2]
When an AML has typical appearances there is essentially no differential. If atypical, especially when fat-poor other lesions to consider include:
- retroperitoneal liposarcoma invading the kidney:
- presence of a large vessel extending into the renal cortex suggestive of AML; liposarcoma is hypovascular
- renal parenchymal defect at the site of tumour contact favours exophytic angiomyolipoma- claw sign
- calcifications suggest liposarcoma
- adrenal myelolipoma
- renal cell carcinoma (RCC)
- may contain fat: lipid necrosis or osseous metaplasia
- oncocytoma: may contain fat
- Wilms tumour: may contain fat
- perirenal fat entrapment / renal junctional parenchymal defect