Tumor lysis syndrome differential diagnosis: Difference between revisions
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::*Type IV [[renal tubular acidosis]] | ::*Type IV [[renal tubular acidosis]] | ||
:*Gordon's syndrome ([[pseudohypoaldosteronism]] type II) | :*Gordon's syndrome ([[pseudohypoaldosteronism]] type II) | ||
*[[Hyperphosphatemia]] | |||
:*[[Acute kidney injury]] | |||
:*[[Hypoparathyroidism]] | |||
:*[[Hypervitaminosis D|Vitamin D]] | |||
:*[[Hypervitaminosis A|vitamin A intoxication]] | |||
:*[[Sarcoidosis]] | |||
:*Immobilization | |||
:*Osteolytic metastases | |||
:*[[Milk-alkali syndrome]] | |||
:*Severe [[hypermagnesemia]] or [[hypomagnesemia]] | |||
:*[[Pseudohypoparathyroidism]] | |||
:*[[Acromegaly]] | |||
:*Extensive cellular injury or necrosis: | |||
::*Crush injury | |||
::*[[Rhabdomyolysis]] | |||
::*[[Hyperthermia]] | |||
<ref name="pmid24359983">{{cite journal| author=Wilson FP, Berns JS| title=Tumor lysis syndrome: new challenges and recent advances. | journal=Adv Chronic Kidney Dis | year= 2014 | volume= 21 | issue= 1 | pages= 18-26 | pmid=24359983 | doi=10.1053/j.ackd.2013.07.001 | pmc=PMC4017246 | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=24359983 }} </ref> | |||
==References== | ==References== | ||
Revision as of 19:30, 29 September 2015
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Mohamad Alkateb, MBBCh [2]
Overview
Tumor lysis syndrome must be differentiated from other diseases that cause hyperuricemia, hyperkalemia, and hyperphosphatemia, such as acute kidney injury.[1]
Differentiating tumor lysis syndrome from other Diseases
Tumor lysis syndrome must be differentiated from other diseases that cause electrolytes disturbance.
- Hereditary hyperuricemia
- Insulin resistance
- Hypertension
- Obesity
- Gout
- Alcoholism
- Renal insufficiency
- Medications:
- Renal insufficiency
- Medications
- ACE inhibitor
- angiotensin receptor blockers
- Potassium-sparing diuretics such asamiloride and spironolactone
- NSAIDs such as ibuprofen andnaproxen
- Ciclosporin
- Tacrolimus
- Antibiotics such as trimethoprim
- Antiparasitic drugs such as pentamidine
- Mineralocorticoid deficiency or resistance, such as:
- Addison's disease
- Aldosterone deficiency
- Some forms of congenital adrenal hyperplasia
- Type IV renal tubular acidosis
- Gordon's syndrome (pseudohypoaldosteronism type II)
- Acute kidney injury
- Hypoparathyroidism
- Vitamin D
- vitamin A intoxication
- Sarcoidosis
- Immobilization
- Osteolytic metastases
- Milk-alkali syndrome
- Severe hypermagnesemia or hypomagnesemia
- Pseudohypoparathyroidism
- Acromegaly
- Extensive cellular injury or necrosis:
- Crush injury
- Rhabdomyolysis
- Hyperthermia
References
- ↑ 1.0 1.1 Wilson FP, Berns JS (2014). "Tumor lysis syndrome: new challenges and recent advances". Adv Chronic Kidney Dis. 21 (1): 18–26. doi:10.1053/j.ackd.2013.07.001. PMC 4017246. PMID 24359983.
- ↑ 2.0 2.1 Wikipedia.https://en.wikipedia.org/wiki/Hyperuricemia