21-hydroxylase deficiency other diagnostic studies: Difference between revisions
No edit summary |
No edit summary |
||
| Line 3: | Line 3: | ||
{{CMG}} {{AE}} {{AAM}} | {{CMG}} {{AE}} {{AAM}} | ||
==Overview== | ==Overview== | ||
Immunohistochemical staining of the adrenal-gland may be used for the diagnosis of congenital adrenal hyperplasia due to 21-hydroxylase deficiency and it shows [[hyperplasia]], poorly defined zonation, and intermingling of the [[chromaffin]] and cortical cells. | |||
==Other Diagnostic Studies== | ==Other Diagnostic Studies== | ||
*Immunohistochemical staining of the adrenal-gland may be used in patients with classic 21-hydroxylase deficiency and it shows: | |||
*[[Hyperplasia]] | |||
*Poorly defined zonation | |||
*Intermingling of the [[chromaffin]] and cortical cells | |||
== References == | == References == | ||
{{Reflist|2}} | {{Reflist|2}} | ||
Revision as of 21:09, 30 September 2015
|
Congenital adrenal hyperplasia due to 21-hydroxylase deficiency Microchapters |
|
Differentiating Congenital adrenal hyperplasia due to 21-hydroxylase deficiency from other Diseases |
|---|
|
Diagnosis |
|
Treatment |
|
Case Studies |
|
21-hydroxylase deficiency other diagnostic studies On the Web |
|
American Roentgen Ray Society Images of 21-hydroxylase deficiency other diagnostic studies |
|
21-hydroxylase deficiency other diagnostic studies in the news |
|
Directions to Hospitals Treating Congenital adrenal hyperplasia due to 21-hydroxylase deficiency |
|
Risk calculators and risk factors for 21-hydroxylase deficiency other diagnostic studies |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Ahmad Al Maradni, M.D. [2]
Overview
Immunohistochemical staining of the adrenal-gland may be used for the diagnosis of congenital adrenal hyperplasia due to 21-hydroxylase deficiency and it shows hyperplasia, poorly defined zonation, and intermingling of the chromaffin and cortical cells.
Other Diagnostic Studies
- Immunohistochemical staining of the adrenal-gland may be used in patients with classic 21-hydroxylase deficiency and it shows:
- Poorly defined zonation
- Intermingling of the chromaffin and cortical cells