21-hydroxylase deficiency other diagnostic studies: Difference between revisions
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==Overview== | ==Overview== | ||
Immunohistochemical staining of the adrenal-gland may be used for the diagnosis of congenital adrenal hyperplasia due to 21 | Immunohistochemical staining of the adrenal-gland may be used for the diagnosis of congenital adrenal hyperplasia due to 21 hydroxylase deficiency and it demonstrates [[hyperplasia]], poorly defined zonation, and intermingling of the [[chromaffin]] and cortical cells. | ||
==Other Diagnostic Studies== | ==Other Diagnostic Studies== | ||
Immunohistochemical staining of the adrenal-gland may be used in patients with classic 21-hydroxylase deficiency and it demonstrates: | Immunohistochemical staining of the adrenal-gland may be used in patients with classic 21-hydroxylase deficiency and it demonstrates: | ||
Revision as of 17:58, 15 October 2015
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Congenital adrenal hyperplasia due to 21-hydroxylase deficiency Microchapters |
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Differentiating Congenital adrenal hyperplasia due to 21-hydroxylase deficiency from other Diseases |
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Diagnosis |
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Treatment |
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Case Studies |
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21-hydroxylase deficiency other diagnostic studies On the Web |
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American Roentgen Ray Society Images of 21-hydroxylase deficiency other diagnostic studies |
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21-hydroxylase deficiency other diagnostic studies in the news |
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Directions to Hospitals Treating Congenital adrenal hyperplasia due to 21-hydroxylase deficiency |
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Risk calculators and risk factors for 21-hydroxylase deficiency other diagnostic studies |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Ahmad Al Maradni, M.D. [2]
Overview
Immunohistochemical staining of the adrenal-gland may be used for the diagnosis of congenital adrenal hyperplasia due to 21 hydroxylase deficiency and it demonstrates hyperplasia, poorly defined zonation, and intermingling of the chromaffin and cortical cells.
Other Diagnostic Studies
Immunohistochemical staining of the adrenal-gland may be used in patients with classic 21-hydroxylase deficiency and it demonstrates:
- Poorly defined zonation
- Intermingling of the chromaffin and cortical cells