Hepatic hemangioma pathophysiology: Difference between revisions
Jump to navigation
Jump to search
Nawal Muazam (talk | contribs) No edit summary |
Nawal Muazam (talk | contribs) No edit summary |
||
Line 9: | Line 9: | ||
===Associated Conditions=== | ===Associated Conditions=== | ||
* May be associated with [[Kasabach-Merritt syndrome]], [[hemolytic anemia]] and consumptive coagulopathy. | * May be associated with [[Kasabach-Merritt syndrome]], [[hemolytic anemia]] and consumptive coagulopathy. | ||
==Gross Pathology== | |||
==Microscopic Pathology== | |||
==References== | ==References== |
Revision as of 19:24, 20 October 2015
Hepatic hemangioma Microchapters |
Diagnosis |
---|
Treatment |
Case Studies |
Hepatic hemangioma pathophysiology On the Web |
American Roentgen Ray Society Images of Hepatic hemangioma pathophysiology |
Risk calculators and risk factors for Hepatic hemangioma pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1];Associate Editor(s)-in-Chief: Nawal Muazam M.D.[2]
Overview
Pathophysiology
- They arise from the endothelial cells that line the blood vessels and consists of multiple, large vascular channels lined by a single layer of endothelial cells and supported by collagenous walls.
- They may be associated with focal nodular hyperplasia.
Associated Conditions
- May be associated with Kasabach-Merritt syndrome, hemolytic anemia and consumptive coagulopathy.