Sporotrichosis pathophysiology: Difference between revisions
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===Pathogenesis=== | ===Pathogenesis=== | ||
The pathophysiology of sporotrichosis depends on the histological subtype. | *The pathophysiology of sporotrichosis depends on the histological subtype. | ||
*The frequently nonspecific histopathology may mimic other granulomatous diseases.<ref name="pmid0.1155/2014/272376">{{cite journal| author=Aoi W, Henry DP, Weinberger MH| title=Evidence for a physiological role of renal sympathetic nerves in adrenergic stimulation of renin release in the rat. | journal=Circ Res | year= 1976 | volume= 38 | issue= 2 | pages= UNKNOWN | pmid=0.1155/2014/272376 | doi= | pmc= | url=http://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=1155 }} </ref> | |||
==References== | ==References== |
Revision as of 16:53, 11 January 2016
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Alison Leibowitz [2]
Overview
Pathophysiology
Transmission
- S. schenckii is usually transmitted to the human host via posttraumatic inoculation. However, sporotrichosis may also develop as a result of spore inhalation, although this mode of transmission is infrequent.
- Modes of transmission either lead to direct inoculation or enable the entry of the fungus.
- Actions, such as handling thorny plants, sphagnum moss, bales of hay, or any plant or plant product that can cause skin trauma, may enable S. schenckii entry.
Pathogenesis
- The pathophysiology of sporotrichosis depends on the histological subtype.
- The frequently nonspecific histopathology may mimic other granulomatous diseases.[1]
References
- ↑ Aoi W, Henry DP, Weinberger MH (1976). "Evidence for a physiological role of renal sympathetic nerves in adrenergic stimulation of renin release in the rat". Circ Res. 38 (2): UNKNOWN. PMID 0.1155/2014/272376 Check
|pmid=
value (help).