Reactive arthritis pathophysiology: Difference between revisions
No edit summary |
m Categories |
||
Line 13: | Line 13: | ||
{{Reflist|2}} | {{Reflist|2}} | ||
[[Category:Rheumatology]] | [[Category:Rheumatology]] | ||
{{WikiDoc Help Menu}} | {{WikiDoc Help Menu}} | ||
{{WikiDoc Sources}} | {{WikiDoc Sources}} |
Revision as of 14:29, 23 June 2016
Reactive arthritis Microchapters |
Diagnosis |
---|
Treatment |
Case Studies |
Reactive arthritis pathophysiology On the Web |
American Roentgen Ray Society Images of Reactive arthritis pathophysiology |
Risk calculators and risk factors forReactive arthritis pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Pathophysiology
It is set off by a preceding infection, the most common of which would be a genital infection with Chlamydia trachomatis in the US.
Reactive arthritis usually manifests about 1-3 weeks after a known infection. The mechanism of interaction between the infecting organism and the host is unknown. Synovial fluid cultures are negative, suggesting that RA is caused either by an over-excited autoimmune response or by bacterial antigens which have somehow become deposited in the joints.