Acute bronchitis pathophysiology: Difference between revisions
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*Following transmission, the bug starts to inoculate the tracheobronchial epithelium. | *Following transmission, the bug starts to inoculate the tracheobronchial epithelium. | ||
*This process leads to inflammation, thickening, and increased mucus production in the airways as shown in figure-1. | *This process leads to inflammation, thickening, and increased mucus production in the airways as shown in figure-1. | ||
< | <gallery> | ||
Image:normal bronchi.jpg|150px | Image:normal bronchi.jpg|150px | ||
Image:acute bronchitis.jpg|150px | Image:acute bronchitis.jpg|150px |
Revision as of 17:15, 8 September 2016
Acute bronchitis Microchapters |
Diagnosis |
Treatment |
Acute bronchitis pathophysiology On the Web |
American Roentgen Ray Society Images of Acute bronchitis pathophysiology |
Risk calculators and risk factors for Acute bronchitis pathophysiology |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]Associate Editor(s)-in-Chief: Seyedmahdi Pahlavani, M.D. [2]
Overview
Pathophysiology
- The causative agent, either viruses or bacteria transmitted via airways to the large and medium size airways tract.
- Following transmission, the bug starts to inoculate the tracheobronchial epithelium.
- This process leads to inflammation, thickening, and increased mucus production in the airways as shown in figure-1.
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