Sandbox:septic arthritis: Difference between revisions

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== Pathophysiology ==
== Pathophysiology ==
Septic arthritis most commonly develop as a result of hematogenous spreading bacteria into the vascular synovial membrane.<ref name="pmid3288326">Klein RS (1988) [https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&retmode=ref&cmd=prlinks&id=3288326 Joint infection, with consideration of underlying disease and sources of bacteremia in hematogenous infection.] ''Clin Geriatr Med'' 4 (2):375-94. PMID: [https://pubmed.gov/3288326 3288326]</ref>
Septic arthritis most commonly develop as a result of hematogenous spreading bacteria into the vascular synovial membrane.<ref name="pmid3288326">Klein RS (1988) [https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&retmode=ref&cmd=prlinks&id=3288326 Joint infection, with consideration of underlying disease and sources of bacteremia in hematogenous infection.] ''Clin Geriatr Med'' 4 (2):375-94. PMID: [https://pubmed.gov/3288326 3288326]</ref>
== Risk Factors ==
Recent history of joint aspiration or local corticosteroid joint injection.<ref name="pmid10554051">Hunter JA, Blyth TH (1999) [https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&retmode=ref&cmd=prlinks&id=10554051 A risk-benefit assessment of intra-articular corticosteroids in rheumatic disorders.] ''Drug Saf'' 21 (5):353-65. PMID: [https://pubmed.gov/10554051 10554051]</ref><ref name="pmid3288326">Klein RS (1988) [https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&retmode=ref&cmd=prlinks&id=3288326 Joint infection, with consideration of underlying disease and sources of bacteremia in hematogenous infection.] ''Clin Geriatr Med'' 4 (2):375-94. PMID: [https://pubmed.gov/3288326 3288326]</ref>


==References==
==References==

Revision as of 19:43, 9 January 2017

Overview

Septic arthritis is a very much important consideration in adults presenting with acute monoarticular arthritis.

Defintion

Historical perspective

Pathophysiology

Septic arthritis most commonly develop as a result of hematogenous spreading bacteria into the vascular synovial membrane.[1]

Risk Factors

Recent history of joint aspiration or local corticosteroid joint injection.[2][1]

References