Bacterial meningitis pathophysiology: Difference between revisions
Jump to navigation
Jump to search
Aysha Aslam (talk | contribs) |
Aysha Aslam (talk | contribs) |
||
| Line 11: | Line 11: | ||
*It may also spread by exchanging throat and respiratory secretions. | *It may also spread by exchanging throat and respiratory secretions. | ||
*Listeria monocytogenes may spread by eating contaminated food. | *Listeria monocytogenes may spread by eating contaminated food. | ||
*Most people are carriers and do not develop the disease. | *Most people are carriers and do not develop the disease. | ||
===Colonization and evasion of host immune response=== | ===Colonization and evasion of host immune response=== | ||
Revision as of 17:52, 10 January 2017
|
Bacterial meningitis Microchapters |
|
Diagnosis |
|
Treatment |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Aysha Anwar, M.B.B.S[2]
Overview
Pathophysiology
Pathogenensis of bacterial meningitis is a complex process which may occur due to imbalance between the host immune response and virulence factors of pathogen causing infection. Following steps may explain the underlying process in a comprehensive way:
Transmission
- Transmission of bacteria causing meningitis may be transmitted by close contact or prolong contact with patient suffering from meningitis such as H. influenza type b and N. meningitides.
- It may also spread by exchanging throat and respiratory secretions.
- Listeria monocytogenes may spread by eating contaminated food.
- Most people are carriers and do not develop the disease.