Staphylococcus aureus infection pathophysiology: Difference between revisions
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__NOTOC__ | __NOTOC__ | ||
{{Staphylococcus aureus infection}} | {{Staphylococcus aureus infection}} | ||
{{CMG}} | {{CMG}}{{AE}}{{FB}} | ||
==Overview== | ==Overview== | ||
==Pathophysiology== | ==Pathophysiology== | ||
Revision as of 16:55, 8 May 2017
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Staphylococcus aureus infection Main page |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]Associate Editor(s)-in-Chief: Fatimo Biobaku M.B.B.S [2]
Overview
Pathophysiology
Staphylococcus aureus is a highly virulent bacteria that has been recognized as a cause of a wide variety of diseases in humans. Several strains of Staphylococcus aureus bacteria exist. The characteristic attribute of a particular strain such as toxins and extracellular factors, invasive properties (such as adherence, biofilm formation, and resistance to phagocytosis), majorly determine the pathogenesis of Staphylococcus aureus infection.[1]
References
- ↑ Chessa D, Ganau G, Mazzarello V (2015). "An overview of Staphylococcus epidermidis and Staphylococcus aureus with a focus on developing countries". J Infect Dev Ctries. 9 (6): 547–50. doi:10.3855/jidc.6923. PMID 26142662.