Primary hyperaldosteronism causes: Difference between revisions
Jump to navigation
Jump to search
No edit summary |
No edit summary |
||
Line 11: | Line 11: | ||
*Extra-adrenal causes | *Extra-adrenal causes | ||
**Ectopic secretion of aldosterone (Ovaries and Kidneys) | **Ectopic secretion of aldosterone (Ovaries and Kidneys) | ||
===Less Common Causes=== | |||
*Familial hyperaldosteronism type I (glucocorticoid-remediable aldosteronism [GRA]) | |||
*Familial hyperaldosteronism II (the familial occurrence of APA or bilateral idiopathic hyperplasia or both) | |||
*Familial hyperaldosteronism type III (associated with the germline mutation in the KCNJ5 potassium channel) | |||
*Pure aldosterone-producing adrenocortical carcinomas | |||
*Unilateral adrenal hyperplasia | |||
Revision as of 17:06, 7 July 2017
Template:Conn syndrome Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Syed Hassan A. Kazmi BSc, MD [2]
Overview
Causes
Common Causes
Causes of Conn's Syndrome may be divided into:
- Adrenal causes:
- Aldosterone-secreting adrenal adenoma (APA-benign tumor, 50-60%)
- Idiopathic hyperaldosteronism (IHA-Bilateral hyperplasia of the adrenal gland, 40-50%)
- Extra-adrenal causes
- Ectopic secretion of aldosterone (Ovaries and Kidneys)
Less Common Causes
- Familial hyperaldosteronism type I (glucocorticoid-remediable aldosteronism [GRA])
- Familial hyperaldosteronism II (the familial occurrence of APA or bilateral idiopathic hyperplasia or both)
- Familial hyperaldosteronism type III (associated with the germline mutation in the KCNJ5 potassium channel)
- Pure aldosterone-producing adrenocortical carcinomas
- Unilateral adrenal hyperplasia