Staphylococcal scalded skin syndrome pathophysiology: Difference between revisions
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Latest revision as of 18:51, 18 September 2017
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Staphylococcal scalded skin syndrome Microchapters |
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Differentiating Staphylococcal scalded skin syndrome from other Diseases |
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Diagnosis |
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Staphylococcal scalded skin syndrome pathophysiology On the Web |
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
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Overview
The syndrome is induced by epidermolytic exotoxins (exfoliatin)[1] A and B, which are released by S. aureus and cause detachment within the epidermal layer; by breaking down tight-junctions. One of the exotoxins is produced by the bacterial chromosome, while the other is produced by a plasmid. (Bacterial plasmids are pieces of self-replicating DNA that often code for secondary characteristics, such as antibiotic resistance, and toxin production.) These exotoxins are proteases that cleave desmoglein-1, which normally holds the granulosum and spinosum layers together.
References