Hemolytic anemia risk factors: Difference between revisions
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==Risk Factors== | ==Risk Factors== | ||
Oxidative stress in the setting of G6PD deficiency:<ref name="pmid24372186">{{cite journal| author=Luzzatto L, Seneca E| title=G6PD deficiency: a classic example of pharmacogenetics with on-going clinical implications. | journal=Br J Haematol | year= 2014 | volume= 164 | issue= 4 | pages= 469-80 | pmid=24372186 | doi=10.1111/bjh.12665 | pmc=4153881 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=24372186 }} </ref> | |||
*Use of primiquine for malaria treatment | |||
*Use of dapsone for PCP or leprosy treatment | |||
*Consumption of fava beans as part of a Mediterranean diet | |||
*Use of sulfa drugs like trimethoprim-sulfamethoxazole for treatment of skin, urinary tract, or other infections | |||
*Use of phenazopyridine for alleviating symptoms of dysuria | |||
*Use of nitrofurantoin for treatment of a urinary tract infection | |||
==References== | ==References== | ||
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
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Overview
Risk Factors
Oxidative stress in the setting of G6PD deficiency:[1]
- Use of primiquine for malaria treatment
- Use of dapsone for PCP or leprosy treatment
- Consumption of fava beans as part of a Mediterranean diet
- Use of sulfa drugs like trimethoprim-sulfamethoxazole for treatment of skin, urinary tract, or other infections
- Use of phenazopyridine for alleviating symptoms of dysuria
- Use of nitrofurantoin for treatment of a urinary tract infection
References
- ↑ Luzzatto L, Seneca E (2014). "G6PD deficiency: a classic example of pharmacogenetics with on-going clinical implications". Br J Haematol. 164 (4): 469–80. doi:10.1111/bjh.12665. PMC 4153881. PMID 24372186.