Gastritis overview: Difference between revisions
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==Risk Factors== | ==Risk Factors== | ||
Common risk factors in the development of Gastritis include alcohol, NSAIDs, cocaine, autoimmune gastritis, Crohn’s disease, HIV/AIDS and bacterial infections such as Helicobacter pylori. | |||
==Screening== | ==Screening== |
Revision as of 10:47, 26 October 2017
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]
Overview
Gastritis is inflammation of the gastric mucosa. The word comes from the Greek gastro- meaning of the stomach and -itis meaning inflammation. Depending on the cause, it may persist acutely or chronically and may coincide with more serious conditions such as atrophy of the stomach.
Historical Perspective
In 1728, a German physician named Georg Ernst Stahl first used the term "gastritis" to describe inflammation of the inner lining of the stomach.
Classification
Gastritis may be classified according to chronicity, into two categories: acute and chronic.
Pathophysiology
Causes
Common causes include: Helicobacter pylori (H. pylori) infection, damage to the stomach lining (such as from NSAIDs), and autoimmune response
Differentiating Gastritis overview from Other Diseases
Gastritis must be differentiated from peptic ulcer disease, gastric cancer, gastroesophageal reflux disease, gastroenteritis, and Crohn's disease.
Epidemiology and Demographics
Risk Factors
Common risk factors in the development of Gastritis include alcohol, NSAIDs, cocaine, autoimmune gastritis, Crohn’s disease, HIV/AIDS and bacterial infections such as Helicobacter pylori.
Screening
Natural History, Complications, and Prognosis
Natural History
Complications
Prognosis
Diagnosis
Diagnostic Criteria
History and Symptoms
Symptoms of gastritis may be silent or manifest as abdominal discomfort, nausea, vomiting, and/or gastrointestinal bleeding.