Budd-Chiari syndrome laboratory findings: Difference between revisions

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Revision as of 19:19, 6 November 2017

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief:

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When Budd-Chiari syndrome is suspected, measurements are made of :
- Liver enzyme levels
- Creatinine
- Urea
- Electrolytes
- LDH
- Liver biopsy is nonspecific but sometimes necessary to differentiate between Budd-Chiari syndrome and other causes of hepatomegaly and ascites, such as galactosemia or Reye's syndrome
- Factor V Leiden and Factor II (prothrombin) mutations, for the presence of antiphospholipid antibodies, is usually tested for patients with primary BCS.
Laboratory findings consistent with the diagnosis of acute and fulminant BCS include:
- Elevated Serum aspartate and alanine aminotransferase levels may be more than five times the upper limit of the normal range.
- Elevated Serum alkaline phosphatase and bilirubin levels, decreased serum albumin level.
Ascitic fluid examination shows:
- Total protein level more than 2.5 g per deciliter
- White blood cells are usually less than 500/μL.
Additional Hematological tests are recommended to evaluate for hypercoagulability.

@@ Line 8: / Line 8: @@
 ==Laboratory Findings==
-When Budd-Chiari syndrome is suspected, measurements are made of :
+*When Budd-Chiari syndrome is suspected, measurements are made of :
+**[[Liver enzyme]] levels
-* [[Liver enzyme]] levels
+**[[Creatinine]]
-* [[Creatinine]]
+**[[Urea]]
-* [[Urea]]
+**[[Electrolyte]]s
-* [[Electrolyte]]s
+**[[lactate dehydrogenase|LDH]]
-* [[lactate dehydrogenase|LDH]]
+**[[Liver biopsy]] is nonspecific but sometimes necessary to differentiate between Budd-Chiari syndrome and other causes of hepatomegaly and ascites, such as [[galactosemia]] or [[Reye's syndrome]]
-* [[Liver biopsy]] is nonspecific but sometimes necessary to differentiate between Budd-Chiari syndrome and other causes of hepatomegaly and ascites, such as [[galactosemia]] or [[Reye's syndrome]]
+**Factor V Leiden and Factor II (prothrombin) mutations, for the presence of antiphospholipid antibodies, is usually tested for patients with primary BCS.
-* Severe hepatic [[necrosis]] and [[lactic acidosis]] may be present as well.  Caudate lobe hypertrophy is often present. The majority of patients have a slower-onset form of Budd-Chiari syndrome. This can be painless.   Patients may progress to [[cirrhosis]] and show the signs of liver failure.
 *Laboratory findings consistent with the diagnosis of acute and fulminant BCS include:
 **Elevated Serum aspartate and alanine aminotransferase levels may be more than five times the upper limit of the normal range.