Pyloric stenosis risk factors: Difference between revisions
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==Overview== | ==Overview== | ||
The most potent risk factors in the development of infantile pyloric stenosis are bottle | The most potent risk factors in the development of infantile pyloric stenosis are bottle feeding, [[caesarean section]] delivery, first-born infant, [[preterm birth]], and exposure to [[Macrolide|macrolides]], [[nitrofurantoin]], [[Penicillin|penicillins]] and [[trimethoprim]]-sulphamethoxazole during [[pregnancy]]. | ||
==Risk Factors== | ==Risk Factors== | ||
=== Risk Factors of infantile pyloric stenosis=== | === Risk Factors of infantile pyloric stenosis=== | ||
The most potent risk factor in the development of infantile pyloric stenosis is male gender. Other risk factors include | The most potent risk factor in the development of infantile pyloric stenosis is male gender. Other risk factors include bottle feeding, caucasian race, [[Caesarean section|cesarean section]] delivery, first born infant, preterm birth and exposure to [[Macrolide|macrolides]], [[nitrofurantoin]], [[Penicillin|penicillins]] and [[trimethoprim]]-sulphamethoxazole during [[pregnancy|pregnancy.]]<ref name="pmid26905846">{{cite journal| author=Nordeng S, Nordeng H, Høye S| title=[Use of antibiotics during pregnancy]. | journal=Tidsskr Nor Laegeforen | year= 2016 | volume= 136 | issue= 4 | pages= 317-21 | pmid=26905846 | doi=10.4045/tidsskr.15.0451 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=26905846 }} </ref> | ||
=== Common Risk Factors of infantile pyloric stenosis === | === Common Risk Factors of infantile pyloric stenosis === | ||
*Male gender | *Male gender | ||
*Caucasian race | *Caucasian race | ||
*Bottle | *Bottle feeding | ||
*[[Caesarean section|Cesarean section]] delivery | *[[Caesarean section|Cesarean section]] delivery | ||
=== Less Common Risk Factors === | === Less Common Risk Factors === | ||
Less common risk factors in the development of infantile pyloric stenosis include: | |||
* First-born infant | |||
*[[Premature birth|Preterm birth]] | |||
*Exposure to [[Macrolide|macrolides]], [[nitrofurantoin]], [[Penicillin|penicillins]] and [[trimethoprim]]-sulphamethoxazole during [[pregnancy]].<ref name="pmid26905846" /> | |||
=== Risk Factors for adult-onset hypertrophic pyloric stenosis (HPS)=== | === Risk Factors for adult-onset hypertrophic pyloric stenosis (HPS)=== | ||
There are no established [[risk factor]] for [[adult]]-onset hypertrophic pyloric stenosis (HPS). | There are no established [[risk factor]] for [[adult]]-onset hypertrophic pyloric stenosis (HPS). | ||
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Mohamadmostafa Jahansouz M.D.[2]
Overview
The most potent risk factors in the development of infantile pyloric stenosis are bottle feeding, caesarean section delivery, first-born infant, preterm birth, and exposure to macrolides, nitrofurantoin, penicillins and trimethoprim-sulphamethoxazole during pregnancy.
Risk Factors
Risk Factors of infantile pyloric stenosis
The most potent risk factor in the development of infantile pyloric stenosis is male gender. Other risk factors include bottle feeding, caucasian race, cesarean section delivery, first born infant, preterm birth and exposure to macrolides, nitrofurantoin, penicillins and trimethoprim-sulphamethoxazole during pregnancy.[1]
Common Risk Factors of infantile pyloric stenosis
- Male gender
- Caucasian race
- Bottle feeding
- Cesarean section delivery
Less Common Risk Factors
Less common risk factors in the development of infantile pyloric stenosis include:
- First-born infant
- Preterm birth
- Exposure to macrolides, nitrofurantoin, penicillins and trimethoprim-sulphamethoxazole during pregnancy.[1]
Risk Factors for adult-onset hypertrophic pyloric stenosis (HPS)
There are no established risk factor for adult-onset hypertrophic pyloric stenosis (HPS).
References
- ↑ 1.0 1.1 Nordeng S, Nordeng H, Høye S (2016). "[Use of antibiotics during pregnancy]". Tidsskr Nor Laegeforen. 136 (4): 317–21. doi:10.4045/tidsskr.15.0451. PMID 26905846.