Spontaneous coronary artery dissection causes: Difference between revisions

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* [[Fibromuscular dysplasia]]
* [[Fibromuscular dysplasia]]
* [[Takotsubo cardiomyopathy]]
* [[Takotsubo cardiomyopathy]]
* [[Pregnancy|Pregnancy-related]] (antepartum, early post-partum, late post-partum, very late post-partum)
* [[Pregnancy|Pregnancy-related]]: antepartum, early post-partum, late post-partum, very late post-partum
* [[Pregnancy|Recurrent pregnancies]]: [[Parity (medicine)|multiparity]] or [[multigravida]]
* [[Pregnancy|Recurrent pregnancies]]: [[Parity (medicine)|multiparity]] or [[multigravida]]
* [[Connective tissue disorder]]: [[Marfan syndrome]], [[Loeys-Dietz syndrome]], [[Ehlers-Danlos syndrome|Ehlers-Danlos syndrome type 4]], [[cystic medial necrosis]], [[alpha-1 antitrypsin deficiency]], [[polycystic kidney disease]]
* [[Connective tissue disorder]]: [[Marfan syndrome]], [[Loeys-Dietz syndrome]], [[Ehlers-Danlos syndrome|Ehlers-Danlos syndrome type 4]], [[cystic medial necrosis]], [[alpha-1 antitrypsin deficiency]], [[polycystic kidney disease]]
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| style="background: #DCDCDC; padding: 5px; text-align: center;" | '''Precipitating stressors'''
| style="background: #DCDCDC; padding: 5px; text-align: center;" | '''Precipitating stressors'''
| style="background: #F5F5F5; padding: 5px;" colspan="2;"|
| style="background: #F5F5F5; padding: 5px;" colspan="2;"|
* Intense exercises including isometric or aerobic activities
* [[exercise|Intense exercises]] including isometric or aerobic activities
* Intense emotional stress
* [[emotional stress|Intense emotional stress]]
* Labor and delivery
* [[Labor|Labor and delivery]]
* Intense Valsava-type activities: retching, vomiting, bowel movement, coughing
* [[Valsava|Valsava-type activities]]: [[retching]], [[vomiting]], [[bowel movement]], [[coughing]]
* Recreational drugs: cocaine, amphetamines, metamphetamines
* [[Recreational drugs]]: [[cocaine]], [[amphetamines]], [[metamphetamines]]
* Intense hormonal therapy: beta-HCG injections, corticosteroids injections
* [[Hormonal therapy]]: [[beta-HCG]], [[corticosteroids]]
|}
|}



Revision as of 19:42, 28 November 2017

Spontaneous Coronary Artery Dissection Microchapters

Home

Overview

Historical Perspective

Classification

Pathophysiology

Causes

Differentiating Spontaneous coronary artery dissection from other Diseases

Epidemiology and Demographics

Risk Factors

Screening

Natural History, Complications and Prognosis

Diagnosis

Diagnostic Approach

History and Symptoms

Physical Examination

Laboratory Findings

Electrocardiogram

Angiography

CT

MRI

Echocardiography

Other Imaging Findings

Other Diagnostic Studies

Treatment

Treatment Approach

Medical Therapy

Percutaneous Coronary Intervention

Surgery

Primary Prevention

Secondary Prevention

Cost-Effectiveness of Therapy

Future or Investigational Therapies

Case Studies

Type 1

Type 2A

Type 2B

Type 3

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Nate Michalak, B.A.

Synonyms and keywords: SCAD

Overview

Spontaneous coronary artery dissection may occur as a result of predisposing factors (e.g., vasculopathy, connective tissue disorder, systemic inflammation) compounded by precipitating stressors (strenuous exercise, emotional stress, medications).

Causes

The exact etiology of spontaneous coronary artery dissection (SCAD) remains elusive and appears to be multifactorial. It has been postulated that the phenotypic manifestation of SCAD may occur as a result of predisposing factors compounded by precipitating stressors.[1] Conditions that have been implicated as a potential cause include:

Potential Cause Condition
Predisposing factors
Precipitating stressors

References

  1. Saw, Jacqueline; Mancini, G.B. John; Humphries, Karin H. (2016). "Contemporary Review on Spontaneous Coronary Artery Dissection". Journal of the American College of Cardiology. 68 (3): 297–312. doi:10.1016/j.jacc.2016.05.034. ISSN 0735-1097.