Spontaneous coronary artery dissection differential diagnosis: Difference between revisions

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==Differential Diagnosis==
==Differential Diagnosis==
While Type 1 SCAD has a pathognomonic appearance on angiography, Type 2 may be and Type 3 typically is indistinguishable from [[atherosclerosis]]. Risk factors and patient history may help determine diagnosis. Intracoronary imaging provides the most objective tool in differentiating between SCAD and [[atheroma]].<ref name="pmid25774346">{{cite journal| author=Yip A, Saw J| title=Spontaneous coronary artery dissection-A review. | journal=Cardiovasc Diagn Ther | year= 2015 | volume= 5 | issue= 1 | pages= 37-48 | pmid=25774346 | doi=10.3978/j.issn.2223-3652.2015.01.08 | pmc=4329168 | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=25774346  }} </ref><ref name="BuccheriPiraino2016">{{cite journal|last1=Buccheri|first1=Dario|last2=Piraino|first2=Davide|last3=Latini|first3=Roberto A.|last4=Andolina|first4=Giuseppe|last5=Cortese|first5=Bernardo|title=Spontaneous coronary artery dissections: A call for action for an underestimated entity|journal=International Journal of Cardiology|volume=214|year=2016|pages=333–335|issn=01675273|doi=10.1016/j.ijcard.2016.03.131}}</ref>
 
Albeit an infrequent condition, spontaneous coronary artery dissection (SCAD) should be included in the differential diagnosis of [[acute coronary syndrome]].
 
 
 
 
Features that favor the diagnosis of SCAD include:
 
* Myocardial infarction in young women (age ≤50)
* Absence of traditional cardiovascular risk factors
* Little or no evidence of coronary atherosclerosis
* Peripartum state
* History of fibromuscular dysplasia
* History of connective tissue disorder or [[systemic inflammation|systemic inflammatory disorder]]:
:* Marfan's syndrome
:* Type 4 Ehlers-Danlos syndrome
:* Loeys-Dietz syndrome
:* Cystic medial necrosis
:* Systemic lupus erythematosus
:* Crohn's disease
:* Ulcerative colitis
:* Polyarteritis nodosa
:* Sarcoidosis
:* Churg-Strauss syndrome
:* Wegener's granulomatosis
:* Rheumatoid arthritis
:* Giant cell arteritis
 
 




Revision as of 16:47, 1 December 2017

Spontaneous Coronary Artery Dissection Microchapters

Home

Overview

Historical Perspective

Classification

Pathophysiology

Causes

Differentiating Spontaneous coronary artery dissection from other Diseases

Epidemiology and Demographics

Risk Factors

Screening

Natural History, Complications and Prognosis

Diagnosis

Diagnostic Approach

History and Symptoms

Physical Examination

Laboratory Findings

Electrocardiogram

Angiography

CT

MRI

Echocardiography

Other Imaging Findings

Other Diagnostic Studies

Treatment

Treatment Approach

Medical Therapy

Percutaneous Coronary Intervention

Surgery

Primary Prevention

Secondary Prevention

Cost-Effectiveness of Therapy

Future or Investigational Therapies

Case Studies

Type 1

Type 2A

Type 2B

Type 3

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Arzu Kalayci, M.D. [2]

Synonyms and keywords: SCAD

Overview

Differential Diagnosis

Albeit an infrequent condition, spontaneous coronary artery dissection (SCAD) should be included in the differential diagnosis of acute coronary syndrome.



Features that favor the diagnosis of SCAD include:

  • Myocardial infarction in young women (age ≤50)
  • Absence of traditional cardiovascular risk factors
  • Little or no evidence of coronary atherosclerosis
  • Peripartum state
  • History of fibromuscular dysplasia
  • History of connective tissue disorder or systemic inflammatory disorder:
  • Marfan's syndrome
  • Type 4 Ehlers-Danlos syndrome
  • Loeys-Dietz syndrome
  • Cystic medial necrosis
  • Systemic lupus erythematosus
  • Crohn's disease
  • Ulcerative colitis
  • Polyarteritis nodosa
  • Sarcoidosis
  • Churg-Strauss syndrome
  • Wegener's granulomatosis
  • Rheumatoid arthritis
  • Giant cell arteritis



References

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