Cyanosis overview: Difference between revisions
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==Causes== | ==Causes== | ||
Cyanosis is commonly caused by respiratory disorders that inhibits oxygen from reaching the alveoli or interrupts its movement across the alveolar interface. It is also seen in a wide variety of cardiac and vascular disorders by mixing oxygenated blood with deoxygenated blood (eg, vascular shunts or intracardiac shunts), structural or vascular alteration in pulmonary blood flow, peripheral vascular diseases, and cardiac decompensation with pulmonary edema or shock. | |||
==Differentiating Hereditary pancreatitis from Other Diseases== | ==Differentiating Hereditary pancreatitis from Other Diseases== | ||
Revision as of 21:46, 20 March 2018
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Cyanosis Microchapters |
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Cyanosis overview On the Web |
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American Roentgen Ray Society Images of Cyanosis overview |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief:
Overview
Historical Perspective
Classification
Pathophysiology
Cyanosis is a bluish or purplish discoloration of skin and mucous membranes. Two mechanisms involved in the development of cyanosis, Systemic arterial oxygen desaturation and increased oxygen absorption by tissues. Cyanosis is evident when arterial oxygen desaturation falls below 85% or the concentration of deoxygenated hemoglobin (Hb) is below 5 gm/dl. Several factors can affect the appearance of cyanosis includes skin pigmentation, Hemoglobin (Hb) levels, oxygen affinity to the hemoglobin (Hb).
Causes
Cyanosis is commonly caused by respiratory disorders that inhibits oxygen from reaching the alveoli or interrupts its movement across the alveolar interface. It is also seen in a wide variety of cardiac and vascular disorders by mixing oxygenated blood with deoxygenated blood (eg, vascular shunts or intracardiac shunts), structural or vascular alteration in pulmonary blood flow, peripheral vascular diseases, and cardiac decompensation with pulmonary edema or shock.