Bell's palsy pathophysiology: Difference between revisions
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==Associated Conditions== | ==Associated Conditions== | ||
# [[Blepharospasm]] | # [[Blepharospasm]]<ref name="pmid11967652">{{cite journal| author=Miwa H, Kondo T, Mizuno Y| title=Bell's palsy-induced blepharospasm. | journal=J Neurol | year= 2002 | volume= 249 | issue= 4 | pages= 452-4 | pmid=11967652 | doi=10.1007/s004150200038 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=11967652 }} </ref> | ||
#* [[Blepharospasm]] has been rarely seen in patients whit with Bell's palsy. | #* [[Blepharospasm]] has been rarely seen in patients whit with Bell's palsy. | ||
#* In most cases [[blepharospasm]] appeared within a month after the onset of Bell's palsy. | #* In most cases [[blepharospasm]] appeared within a month after the onset of Bell's palsy. |
Revision as of 21:12, 26 April 2018
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] ; Associate Editor(s)-in-Chief: Mohamadmostafa Jahansouz M.D.[2]
Overview
The exact pathogenesis of [disease name] is not fully understood.
OR
It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
OR
[Pathogen name] is usually transmitted via the [transmission route] route to the human host.
OR
Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
OR
[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
OR
The progression to [disease name] usually involves the [molecular pathway].
OR
The pathophysiology of [disease/malignancy] depends on the histological subtype.
Pathophysiology
Pathogenesis
- It is understood that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
- [Pathogen name] is usually transmitted via the [transmission route] route to the human host.
- Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
- [Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
- The progression to [disease name] usually involves the [molecular pathway].
- The pathophysiology of [disease/malignancy] depends on the histological subtype.
Genetics
- There is no established association between genetic factors and Bell's palsy.
Associated Conditions
- Blepharospasm[1]
- Blepharospasm has been rarely seen in patients whit with Bell's palsy.
- In most cases blepharospasm appeared within a month after the onset of Bell's palsy.
Gross Pathology
- On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].
Microscopic Pathology
- On microscopic histopathological analysis, thickened perineurium, infiltrates of inflammatory cells between nerve bundles and around blood vessels are characteristic findings of Bell’s palsy.
- It appears that the histology of the facial nerve in Bell's palsy is similar to Herpes Zoster infection, suggestive of an infectious cause.
References
- ↑ Miwa H, Kondo T, Mizuno Y (2002). "Bell's palsy-induced blepharospasm". J Neurol. 249 (4): 452–4. doi:10.1007/s004150200038. PMID 11967652.