Bell's palsy pathophysiology: Difference between revisions
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#* [[Blepharospasm]] has been rarely seen in patients whit with Bell's palsy. | #* [[Blepharospasm]] has been rarely seen in patients whit with Bell's palsy. | ||
#* In most cases [[blepharospasm]] appeared within a month after the onset of Bell's palsy. | #* In most cases [[blepharospasm]] appeared within a month after the onset of Bell's palsy. | ||
2. [[Herpes zoster]] infection | 2. [[Herpes zoster]] infection<ref name="pmid11096766">{{cite journal| author=Morrow MJ| title=Bell's Palsy and Herpes Zoster Oticus. | journal=Curr Treat Options Neurol | year= 2000 | volume= 2 | issue= 5 | pages= 407-416 | pmid=11096766 | doi= | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=11096766 }} </ref> | ||
==Microscopic Pathology== | ==Microscopic Pathology== | ||
Revision as of 21:37, 26 April 2018
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] ; Associate Editor(s)-in-Chief: Mohamadmostafa Jahansouz M.D.[2]
Overview
The exact pathogenesis of [disease name] is not fully understood.
OR
It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
OR
[Pathogen name] is usually transmitted via the [transmission route] route to the human host.
OR
Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
OR
[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
OR
The progression to [disease name] usually involves the [molecular pathway].
OR
The pathophysiology of [disease/malignancy] depends on the histological subtype.
Pathophysiology
Pathogenesis
- The exact pathophysiology of Bell' palsy is not known.[1]
Genetics
- There is no established association between genetic factors and Bell's palsy.
- Hereditary components may play a role in familial recurrent Bell's palsy.[2]
Associated Conditions
- Blepharospasm[3]
- Blepharospasm has been rarely seen in patients whit with Bell's palsy.
- In most cases blepharospasm appeared within a month after the onset of Bell's palsy.
2. Herpes zoster infection[4]
Microscopic Pathology
- On microscopic histopathological analysis, thickened perineurium, infiltrates of inflammatory cells between nerve bundles and around blood vessels are characteristic findings of Bell’s palsy.[5]
- It appears that the histology of the facial nerve in Bell's palsy is similar to Herpes Zoster infection, suggestive of an infectious cause.[4]
References
- ↑ Somasundara D, Sullivan F (2017). "Management of Bell's palsy". Aust Prescr. 40 (3): 94–97. doi:10.18773/austprescr.2017.030. PMC 5478391. PMID 28798513.
- ↑ Qin D, Ouyang Z, Luo W (2009). "Familial recurrent Bell's palsy". Neurol India. 57 (6): 783–4. doi:10.4103/0028-3886.59478. PMID 20139511.
- ↑ Miwa H, Kondo T, Mizuno Y (2002). "Bell's palsy-induced blepharospasm". J Neurol. 249 (4): 452–4. doi:10.1007/s004150200038. PMID 11967652.
- ↑ 4.0 4.1 Morrow MJ (2000). "Bell's Palsy and Herpes Zoster Oticus". Curr Treat Options Neurol. 2 (5): 407–416. PMID 11096766.
- ↑ Liston SL, Kleid MS (1989). "Histopathology of Bell's palsy". Laryngoscope. 99 (1): 23–6. doi:10.1288/00005537-198901000-00006. PMID 2642582.