Acute tubular necrosis history and symptoms: Difference between revisions
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*Symptoms of [disease name] include [symptom 1], [symptom 2], and [symptom 3]. | *Symptoms of [disease name] include [symptom 1], [symptom 2], and [symptom 3]. | ||
===History=== | ===History=== | ||
Obtaining the | Obtaining the history is the most important aspect of making a [[diagnosis]] of acute tubular necrosis. It provides insight into the [[Etiology|cause]], precipitating factors, and associated comorbid conditions leading to decreased [[Kidney|renal]] blood flow and acute [[Renal tubules|tubular]]/[[kidney]] [[injury]]. A complete [[History and Physical examination|history]] will help determine the correct [[therapy]] and the [[prognosis]]. Specific areas of focus when obtaining the history are outlined below:<ref name="pmid25795495">{{cite journal |vauthors=Alobaidi R, Basu RK, Goldstein SL, Bagshaw SM |title=Sepsis-associated acute kidney injury |journal=Semin. Nephrol. |volume=35 |issue=1 |pages=2–11 |date=January 2015 |pmid=25795495 |pmc=4507081 |doi=10.1016/j.semnephrol.2015.01.002 |url=}}</ref><ref name="pmid16949378">{{cite journal |vauthors=McCullough PA, Adam A, Becker CR, Davidson C, Lameire N, Stacul F, Tumlin J |title=Risk prediction of contrast-induced nephropathy |journal=Am. J. Cardiol. |volume=98 |issue=6A |pages=27K–36K |date=September 2006 |pmid=16949378 |doi=10.1016/j.amjcard.2006.01.022 |url=}}</ref><ref name="pmid16011448">{{cite journal |vauthors=Perazella MA |title=Drug-induced nephropathy: an update |journal=Expert Opin Drug Saf |volume=4 |issue=4 |pages=689–706 |date=July 2005 |pmid=16011448 |doi=10.1517/14740338.4.4.689 |url=}}</ref><ref name="pmid28580076">{{cite journal |vauthors=Park JT |title=Postoperative acute kidney injury |journal=Korean J Anesthesiol |volume=70 |issue=3 |pages=258–266 |date=June 2017 |pmid=28580076 |pmc=5453887 |doi=10.4097/kjae.2017.70.3.258 |url=}}</ref><ref name="pmid28486690">{{cite journal |vauthors=Ngajilo D, Ehrlich R |title=Rhabdomyolysis with acute tubular necrosis following occupational inhalation of thinners |journal=Occup Med (Lond) |volume=67 |issue=5 |pages=401–403 |date=July 2017 |pmid=28486690 |doi=10.1093/occmed/kqx048 |url=}}</ref> | ||
* History of recent [[surgery]] | * History of recent [[surgery]] | ||
* [[Sepsis]] | * [[Sepsis]] | ||
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===Symptoms=== | ===Symptoms=== | ||
Common symptoms of acute tubular necrosis include: Symptoms are mostly related to | Common [[Symptom|symptoms]] of acute tubular necrosis include: Symptoms are mostly related to decreased renal perfusion and acute [[kidney]] damage. | ||
* Decreased or absent urine output | * Decreased or absent [[urine]] output | ||
* Lassitude | * [[Fatigue|Lassitude]] | ||
* Excessive thirst | * Excessive [[thirst]] | ||
* Generalised edema | * Generalised [[edema]] | ||
* Postural dizziness | * Postural [[dizziness]] | ||
* Tachycardia | * [[Tachycardia|Rapid pulses]] | ||
* Muscle cramps | * [[Cramp|Muscle cramps]] | ||
* Confusion | * [[Confusion]] | ||
* Altered mental status | * [[Altered mental status]] | ||
* | * [[Stupor]] or [[coma]] | ||
Revision as of 20:34, 1 May 2018
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief: Chandrakala Yannam, MD [2]
Overview
The majority of patients with [disease name] are asymptomatic.
OR
The hallmark of [disease name] is [finding]. A positive history of [finding 1] and [finding 2] is suggestive of [disease name]. The most common symptoms of [disease name] include [symptom 1], [symptom 2], and [symptom 3]. Common symptoms of [disease] include [symptom 1], [symptom 2], and [symptom 3]. Less common symptoms of [disease name] include [symptom 1], [symptom 2], and [symptom 3].
History and Symptoms
- The majority of patients with [disease name] are asymptomatic.
OR
- The hallmark of [disease name] is [finding]. A positive history of [finding 1] and [finding 2] is suggestive of [disease name]. The most common symptoms of [disease name] include [symptom 1], [symptom 2], and [symptom 3].
- Symptoms of [disease name] include [symptom 1], [symptom 2], and [symptom 3].
History
Obtaining the history is the most important aspect of making a diagnosis of acute tubular necrosis. It provides insight into the cause, precipitating factors, and associated comorbid conditions leading to decreased renal blood flow and acute tubular/kidney injury. A complete history will help determine the correct therapy and the prognosis. Specific areas of focus when obtaining the history are outlined below:[1][2][3][4][5]
- History of recent surgery
- Sepsis
- Profound hypotension
- Severe volume loss (eg, diarrhea, vomiting, bleeding and third-spacing)
- Medication history (eg, exposure to nephrotoxic drugs and radiocontrast substances)
- Tissue injury involving muscles (rabdomyolysis)
- Associated conditions: History of pre-existing medical conditions such as multiple myeloma and diabetes mellitus may cause a decreased renal function
Symptoms
Common symptoms of acute tubular necrosis include: Symptoms are mostly related to decreased renal perfusion and acute kidney damage.
- Decreased or absent urine output
- Lassitude
- Excessive thirst
- Generalised edema
- Postural dizziness
- Rapid pulses
- Muscle cramps
- Confusion
- Altered mental status
- Stupor or coma
References
- ↑ Alobaidi R, Basu RK, Goldstein SL, Bagshaw SM (January 2015). "Sepsis-associated acute kidney injury". Semin. Nephrol. 35 (1): 2–11. doi:10.1016/j.semnephrol.2015.01.002. PMC 4507081. PMID 25795495.
- ↑ McCullough PA, Adam A, Becker CR, Davidson C, Lameire N, Stacul F, Tumlin J (September 2006). "Risk prediction of contrast-induced nephropathy". Am. J. Cardiol. 98 (6A): 27K–36K. doi:10.1016/j.amjcard.2006.01.022. PMID 16949378.
- ↑ Perazella MA (July 2005). "Drug-induced nephropathy: an update". Expert Opin Drug Saf. 4 (4): 689–706. doi:10.1517/14740338.4.4.689. PMID 16011448.
- ↑ Park JT (June 2017). "Postoperative acute kidney injury". Korean J Anesthesiol. 70 (3): 258–266. doi:10.4097/kjae.2017.70.3.258. PMC 5453887. PMID 28580076.
- ↑ Ngajilo D, Ehrlich R (July 2017). "Rhabdomyolysis with acute tubular necrosis following occupational inhalation of thinners". Occup Med (Lond). 67 (5): 401–403. doi:10.1093/occmed/kqx048. PMID 28486690.