Interstitial nephritis: Difference between revisions
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==[[Interstitial nephritis classification|Classification]]== | ==[[Interstitial nephritis classification|Classification]]== | ||
Tubulointerstitial nephritis (TIN) commonly attributes to inflammatory | Tubulointerstitial nephritis (TIN) commonly attributes to inflammatory responses of the kidney with involvement the interstitium and tubules. In this condition the glomeruli are intact or may affect in the late stage of disease. | ||
According to clinical manifestations and the inflammatory process, TIN, in spite of the etiologic agent, can be divided into acute and chronic categories. | |||
==[[Interstitial nephritis pathophysiology|Pathophysiology]]== | ==[[Interstitial nephritis pathophysiology|Pathophysiology]]== | ||
Revision as of 15:31, 18 July 2018
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Interstitial nephritis Microchapters |
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Interstitial nephritis On the Web |
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American Roentgen Ray Society Images of Interstitial nephritis |
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Risk calculators and risk factors for Interstitial nephritis |
Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor(s)-in-Chief: Mohsen Basiri M.D.
Synonyms and keywords: Tubulo-interstitial nephritis
Overview
Two main diseases involve the renal tubules are: Acute tubular necrosis due to Ischemic or toxic injury for more about ATN click here; and tubulointerstitial nephritis with Inflammatory involvement of tubules and interstitium and its consequent reactions.
Since some cases of TIN are due to bacterial infection, and the renal pelvis is deeply involved, therefore pyelonephritis is term describes this condition; and In general ,the term interstitial nephritis is used for TIN that are owing to nonbacterial causes of tubular injury such as drugs, viral infections,autoimmune systemic diseases in which these condition mechanism of damage is due to inflammatory responses not direct damage.
Historical Perspective
Classification
Tubulointerstitial nephritis (TIN) commonly attributes to inflammatory responses of the kidney with involvement the interstitium and tubules. In this condition the glomeruli are intact or may affect in the late stage of disease.
According to clinical manifestations and the inflammatory process, TIN, in spite of the etiologic agent, can be divided into acute and chronic categories.
Pathophysiology
Causes
Differentiating Interstitial nephritis from other Diseases
Epidemiology and Demographics
Risk Factors
Natural History, Complications and Prognosis
Diagnosis
History and Symptoms | Physical Examination | Laboratory Findings | KUB X Ray | CT | MRI | Biopsy and Ultrasound | Other Imaging Findings | Other Diagnostic Studies
Treatment
Medical Therapy | Primary Prevention | Secondary Prevention | Cost-Effectiveness of Therapy | Future or Investigational Therapies