Pseudotumor cerebri pathophysiology: Difference between revisions

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Revision as of 11:57, 27 July 2018

Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1]; Associate Editor(s)-in-Chief:

Overview

Pathophysiology

Pathogenesis

The exact pathogenesis of pseudotumor cerebri is not completely understood. Idiopathic intracranial hypertension or pseudotumor cerebri is defined by the symptoms of increased intracranial pressure without any evidence of tumor.(1uptodate)

Any theory regarding the pathophysiology of this disease should explain the following statements:

High incidence rate in women with childbearing age
Reduced conductance to CSF outflow(25 wall)
Normal ventricle size and lack of hydrocephalus(26 wall)
No evidence of cerebral edema(27 wall)

Genetics

[Disease name] is transmitted in [mode of genetic transmission] pattern.

OR

Genes involved in the pathogenesis of [disease name] include:

[Gene1]
[Gene2]
[Gene3]

OR

The development of [disease name] is the result of multiple genetic mutations such as:

[Mutation 1]
[Mutation 2]
[Mutation 3]

Associated Conditions

Gross Pathology

On gross pathology, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].

Microscopic Pathology

On microscopic histopathological analysis, [feature1], [feature2], and [feature3] are characteristic findings of [disease name].

References

Template:WH Template:WS

@@ Line 4: / Line 4: @@
 {{CMG}}; {{AE}}
 ==Overview==
-The exact pathogenesis of [disease name] is not fully understood.
-OR
-It is thought that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
-OR
-[Pathogen name] is usually transmitted via the [transmission route] route to the human host.
-OR
-Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
-OR
-[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
-OR
-The progression to [disease name] usually involves the [molecular pathway].
-OR
-The pathophysiology of [disease/malignancy] depends on the histological subtype.
 ==Pathophysiology==
-===Physiology===
+===Pathogenesis===
-The normal physiology of [name of process] can be understood as follows:
+The exact pathogenesis of pseudotumor cerebri is not completely understood. Idiopathic intracranial hypertension or pseudotumor cerebri is defined by the symptoms of increased intracranial pressure without any evidence of tumor.(1uptodate)
-===Pathogenesis===
+Any theory regarding the pathophysiology of this disease should explain the following statements:
-*The exact pathogenesis of [disease name] is not completely understood.
+* High incidence rate in women with childbearing age
-OR
+* Reduced conductance to CSF outflow(25 wall)
-*It is understood that [disease name] is the result of / is mediated by / is produced by / is caused by either [hypothesis 1], [hypothesis 2], or [hypothesis 3].
+* Normal ventricle size and lack of hydrocephalus(26 wall)
-*[Pathogen name] is usually transmitted via the [transmission route] route to the human host.
+* No evidence of cerebral edema(27 wall)
-*Following transmission/ingestion, the [pathogen] uses the [entry site] to invade the [cell name] cell.
-*[Disease or malignancy name] arises from [cell name]s, which are [cell type] cells that are normally involved in [function of cells].
-*The progression to [disease name] usually involves the [molecular pathway].
-*The pathophysiology of [disease/malignancy] depends on the histological subtype.
 ==Genetics==