Tricuspid atresia natural history, complications and prognosis: Difference between revisions
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===Prognosis=== | ===Prognosis=== | ||
*The prognosis is poor in untreated patients. | *The prognosis is poor in untreated patients.<ref name="pmid11096554">{{cite journal| author=Rao PS| title=Tricuspid Atresia. | journal=Curr Treat Options Cardiovasc Med | year= 2000 | volume= 2 | issue= 6 | pages= 507-520 | pmid=11096554 | doi=10.1007/s11936-000-0046-6 | pmc= | url=https://www.ncbi.nlm.nih.gov/entrez/eutils/elink.fcgi?dbfrom=pubmed&tool=sumsearch.org/cite&retmode=ref&cmd=prlinks&id=11096554 }} </ref> | ||
==References== | ==References== |
Revision as of 18:33, 2 April 2020
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Editor-In-Chief: C. Michael Gibson, M.S., M.D. [1] Associate Editor-In-Chief: Keri Shafer, M.D. [2] Priyamvada Singh, MBBS [3] Assistant Editor-In-Chief: Kristin Feeney, B.S. [4]
Overview
Natural History, Complications, and Prognosis
Natural History
Complications
- Irregular, fast heart rhythms (arrhythmias)
- Chronic diarrhea (from a disease called protein losing enteropathy)
- Heart failure
- Fluid in the abdomen (ascites) and in the lungs (pleural effusion)
- Blockage of the artificial shunt
- Strokes and other neurological complications
- Sudden death
Prognosis
- The prognosis is poor in untreated patients.[1]
References
- ↑ Rao PS (2000). "Tricuspid Atresia". Curr Treat Options Cardiovasc Med. 2 (6): 507–520. doi:10.1007/s11936-000-0046-6. PMID 11096554.